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Dioxin exposure and non-malignant health effects: a mortality study

Dioxin exposure and non-malignant health effects: a mortality study

http://www.wikidata.org/entity/Q35560647

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Evaluation of diabetes mellitus, serum glucose, and thyroid function among United States workers exposed to 2,3,7,8-tetrachlorodibenzo- p-dioxin Dioxin and diabetes mellitus: an analysis of the combined NIOSH and Ranch Hand data Human health effects after exposure to 2,3,7,8-TCDD The Seveso studies on early and long-term effects of dioxin exposure: a review Understanding the human health effects of chemical mixtures.Hazardous air pollutants and asthma.Environmental contaminants as etiologic factors for diabetes Exposure assessment to dioxins from the use of tampons and diapers Hazardous waste sites and stroke in New York State.Prevalence of self-reported diabetes and exposure to organochlorine pesticides among Mexican Americans: Hispanic health and nutrition examination survey, 1982-1984 High serum PCBs are associated with elevation of serum lipids and cardiovascular disease in a Native American population Induction of oxidative stress responses by dioxin and other ligands of the aryl hydrocarbon receptor Exposure to Persistent Organic Pollutants Increases Hospitalization Rates for Myocardial Infarction with Comorbid Hypertension Endocrine-disrupting chemicals: associated disorders and mechanisms of action Perspectives on the potential involvement of the AH receptor-dioxin axis in cardiovascular disease A cross-sectional analysis of dioxins and health effects in municipal and private waste incinerator workers in Japan Persistent organic pollutants as risk factors for type 2 diabetes Increased rate of hospitalization for diabetes and residential proximity of hazardous waste sites Dioxin (2,3,7,8-tetrachlorodibenzo-p-dioxin) enhances triggered afterdepolarizations in rat ventricular myocytes Association between serum concentrations of persistent organic pollutants and self-reported cardiovascular disease prevalence: results from the National Health and Nutrition Examination Survey, 1999-2002 Diabetes in relation to serum levels of polychlorinated biphenyls and chlorinated pesticides in adult Native Americans Allelic variation and environmental lead exposure in urban children Polychlorinated biphenyl-induced VCAM-1 expression is attenuated in aortic endothelial cells isolated from caveolin-1 deficient mice.Association between serum concentrations of persistent organic pollutants and prevalence of newly diagnosed hypertension: results from the National Health and Nutrition Examination Survey 1999-2002.Pesticide use and incident diabetes among wives of farmers in the Agricultural Health Study.Chloracne, goiter, arthritis, and anemia after polychlorinated biphenyl poisoning: 14-year follow-Up of the Taiwan Yucheng cohort.Basis for a proposed reference dose (RfD) for dioxin of 1-10 pg/kg-day: a weight of evidence evaluation of the human and animal studies.2,3,7,8-Tetrachlorodibenzo-p-dioxin-induced MUC5AC expression: aryl hydrocarbon receptor-independent/EGFR/ERK/p38-dependent SP1-based transcription TCDD inhibits heart regeneration in adult zebrafish Environmental contaminants as risk factors for developing diabetes.Diabetes Prevalence in Relation to Serum Concentrations of Polychlorinated Biphenyl (PCB) Congener Groups and Three Chlorinated Pesticides in a Native American Population Differences in the clinical manifestations and short-term prognosis of acute cerebral infarction after exposure to Agent Orange.Environmental pollutants and type 2 diabetes: a review of mechanisms that can disrupt beta cell function.The role of endogenous aryl hydrocarbon receptor signaling in cardiovascular physiology NADPH oxidase and lipid raft-associated redox signaling are required for PCB153-induced upregulation of cell adhesion molecules in human brain endothelial cells.2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces calcium influx through T-type calcium channel and enhances lysosomal exocytosis and insulin secretion in INS-1 cells.Heart malformation is an early response to TCDD in embryonic zebrafish.Developmental exposure to the potent aryl hydrocarbon receptor agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin Impairs the cell-mediated immune response to infection with influenza a virus, but enhances elements of innate immunity.Activation of the aryl hydrocarbon receptor increases pulmonary neutrophilia and diminishes host resistance to influenza A virus.3-Methylcholanthrene/Aryl-Hydrocarbon Receptor-Mediated Hypertension Through eNOS Inactivation.

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P2860

Dioxin exposure and non-malignant health effects: a mortality study

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1998 nî lūn-bûn

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1998年论文

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1998年论文

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1998年论文

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Occupational and Environmental Medicine

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Dioxin exposure and non-malignant health effects: a mortality study

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Consonni D

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Guercilena S

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Pesatori AC

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Turrini D

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P2860

Evaluation of serum lipid concentrations among U.S. workers exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin

Cancer mortality in workers exposed to 2,3,7,8-tetrachlorodibenzo- p-dioxin

Indirect methods of assessing the effects of tobacco use in occupational studies

Hepatic and gastrointestinal effects in an occupational cohort exposed to 2,3,7,8-tetrachlorodibenzo-para-dioxin

Medical and morbidity surveillance findings among employees potentially exposed to TCDD.

Tetrachlorodibenzodioxin: a survey of subjects ten years after exposure.

Lipid abnormalities in workers exposed to dioxin

Cause specific mortality and cancer incidence among employees exposed to 2,3,7,8-TCDD after a 1953 reactor accident.

Health status of workers with past exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin in the manufacture of 2,4,5-trichlorophenoxyacetic acid: comparison of findings with and without chloracne.

Results of a two-year chronic toxicity and oncogenicity study of 2,3,7,8-tetrachlorodibenzo-p-dioxin in rats.

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126-131

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2

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55

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Pier Alberto Bertazzi

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1998-02-01T00:00:00Z

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13670756

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1757550

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