Functional muscle ischemia in neuronal nitric oxide synthase-deficient skeletal muscle of children with Duchenne muscular dystrophy.
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Respiratory muscle training in children and adults with neuromuscular diseaseGene expression comparison of biopsies from Duchenne muscular dystrophy (DMD) and normal skeletal muscleThe Dietary Supplement Protandim® Decreases Plasma Osteopontin and Improves Markers of Oxidative Stress in Muscular Dystrophy Mdx MiceSildenafil and cardiomyocyte-specific cGMP signaling prevent cardiomyopathic changes associated with dystrophin deficiencyRecent advances in innovative therapeutic approaches for Duchenne muscular dystrophy: from discovery to clinical trialsNeuronal Nitric Oxide Synthase in Vascular Physiology and DiseasesVascular endothelial dysfunction and pharmacological treatmentCardiovascular Reflexes Activity and Their Interaction during ExerciseThe potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophyAutonomic, locomotor and cardiac abnormalities in a mouse model of muscular dystrophy: targeting the renin-angiotensin systemPrimary role of functional ischemia, quantitative evidence for the two-hit mechanism, and phosphodiesterase-5 inhibitor therapy in mouse muscular dystrophyThe burrowing behavior of the nematode Caenorhabditis elegans: a new assay for the study of neuromuscular disordersNutraceuticals and Their Potential to Treat Duchenne Muscular Dystrophy: Separating the Credible from the ConjectureMuscle fatigue, nNOS and muscle fiber atrophy in limb girdle muscular dystrophyAssociation of neuronal nitric oxide synthase (nNOS) with alpha1-syntrophin at the sarcolemmaNeuronal nitric oxide synthase is dislocated in type I fibers of myalgic muscle but can recover with physical exercise trainingTreatment with a nitric oxide-donating NSAID alleviates functional muscle ischemia in the mouse model of Duchenne muscular dystrophyCardiac involvement in patients with muscular dystrophies: magnetic resonance imaging phenotype and genotypic considerations.Blockade of ActRIIB signaling triggers muscle fatigability and metabolic myopathy.The Subcellular Localisation of Neuronal Nitric Oxide Synthase Determines the Downstream Effects of NO on Myocardial FunctionChronic hypoxia impairs muscle function in the Drosophila model of Duchenne's muscular dystrophy (DMD).Use of imaging biomarkers to assess perfusion and glucose metabolism in the skeletal muscle of dystrophic mice.Inhibition of alpha-adrenergic vasoconstriction in exercising human thigh muscles.Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthaseExogenous NO administration and alpha-adrenergic vasoconstriction in human limbs.Functional deficits in nNOSmu-deficient skeletal muscle: myopathy in nNOS knockout miceDystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesHigh levels of sarcospan are well tolerated and act as a sarcolemmal stabilizer to address skeletal muscle and pulmonary dysfunction in DMD.Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophyGolgi and sarcolemmal neuronal NOS differentially regulate contraction-induced fatigue and vasoconstriction in exercising mouse skeletal muscleMyogenin regulates exercise capacity but is dispensable for skeletal muscle regeneration in adult mdx mice.Sildenafil increases muscle protein synthesis and reduces muscle fatigue.Sarcolemmal nNOS anchoring reveals a qualitative difference between dystrophin and utrophin.PDE5 inhibition alleviates functional muscle ischemia in boys with Duchenne muscular dystrophy.Neuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membraneCaspase-12 ablation preserves muscle function in the mdx mouse.Genetic association analyses of nitric oxide synthase genes and neural tube defects vary by phenotypeNeuronal nitric oxide synthase and human vascular regulationRespiratory care of the patient with Duchenne muscular dystrophy: ATS consensus statement.Aminoglycoside-induced mutation suppression (stop codon readthrough) as a therapeutic strategy for Duchenne muscular dystrophy
P2860
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P2860
Functional muscle ischemia in neuronal nitric oxide synthase-deficient skeletal muscle of children with Duchenne muscular dystrophy.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@ast
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@en
type
label
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@ast
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@en
prefLabel
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@ast
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@en
P2093
P2860
P356
P1476
Functional muscle ischemia in ...... h Duchenne muscular dystrophy.
@en
P2093
B Chavoshan
G D Thomas
R G Victor
S A Harris
S T Iannaccone
P2860
P304
13818-13823
P356
10.1073/PNAS.250379497
P407
P577
2000-12-01T00:00:00Z