Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy. - Wikidata - awgldk - TriplyDB

Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy.

Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q35578778

about

Cardiac regulation by phosphoinositide 3-kinases and PTEN BCR Signaling Inhibitors: an Overview of Toxicities Associated with Ibrutinib and Idelalisib in Patients with Chronic Lymphocytic Leukemia Phosphoinositide 3-kinase: friend and foe in cardiovascular disease Using exercise to measure and modify cardiac function Experimental studies of the molecular pathways regulated by exercise and resveratrol in heart, skeletal muscle and the vasculature Myosin light chain phosphorylation is critical for adaptation to cardiac stress.Soluble epoxide hydrolase plays an essential role in angiotensin II-induced cardiac hypertrophy Computer-aided targeting of the PI3K/Akt/mTOR pathway: toxicity reduction and therapeutic opportunities CpG-ODN attenuates pathological cardiac hypertrophy and heart failure by activation of PI3Kα-Akt signaling Association of PI3K-Akt signaling pathway with digitalis-induced hypertrophy of cardiac myocytes Induction of manganese superoxide dismutase by nuclear translocation and activation of SIRT1 promotes cell survival in chronic heart failure Vinculin directly binds zonula occludens-1 and is essential for stabilizing connexin-43-containing gap junctions in cardiac myocytes Insulin Signaling and Heart Failure.Protein Kinases as Drug Development Targets for Heart Disease Therapy Genome-wide expression patterns in physiological cardiac hypertrophy.Effect of lithium on ventricular remodelling in infarcted rats via the Akt/mTOR signalling pathways Exenatide improves glucose homeostasis and prolongs survival in a murine model of dilated cardiomyopathy.Inhibition of the insulin-like growth factor-1 receptor (IGF1R) tyrosine kinase as a novel cancer therapy approach Concomitant low-dose doxorubicin treatment and exercise MicroRNAs 29 are involved in the improvement of ventricular compliance promoted by aerobic exercise training in rats EPC-derived microvesicles protect cardiomyocytes from Ang II-induced hypertrophy and apoptosis Modulation of anthracycline-induced cardiotoxicity by aerobic exercise in breast cancer: current evidence and underlying mechanisms.Exercise protects against chronic β-adrenergic remodeling of the heart by activation of endothelial nitric oxide synthase.Phosphoinositide 3-kinase (PI3K(p110alpha)) directly regulates key components of the Z-disc and cardiac structure.Endurance training or beta-blockade can partially block the energy metabolism remodeling taking place in experimental chronic left ventricle volume overload Enhanced cardiac PI3Kα signalling mitigates arrhythmogenic electrical remodelling in pathological hypertrophy and heart failure Moderate-intensity treadmill exercise training decreases murine cardiomyocyte cross-sectional area.Transcription factor GATA4 is activated but not required for insulin-like growth factor 1 (IGF1)-induced cardiac hypertrophy Combined deep microRNA and mRNA sequencing identifies protective transcriptomal signature of enhanced PI3Kα signaling in cardiac hypertrophy.Cancer Therapy Targeting the HER2-PI3K Pathway: Potential Impact on the Heart.Distinctive ERK and p38 signaling in remote and infarcted myocardium during post-MI remodeling in the mouse.Influence of Physical Activity on Hypertension and Cardiac Structure and Function Therapeutic inhibition of the miR-34 family attenuates pathological cardiac remodeling and improves heart function Perturbations in the gene regulatory pathways controlling mitochondrial energy production in the failing heart.The potential role of aerobic exercise to modulate cardiotoxicity of molecularly targeted cancer therapeutics.Inhibition of miR-154 Protects Against Cardiac Dysfunction and Fibrosis in a Mouse Model of Pressure Overload Insulin-like growth factor 1 prevents diastolic and systolic dysfunction associated with cardiomyopathy and preserves adrenergic sensitivity "Canonical and non-canonical actions of GRK5 in the heart"Phosphatidylinositol-3,4,5-triphosphate and cellular signaling: implications for obesity and diabetes.Differential Role of G Protein-Coupled Receptor Kinase 5 in Physiological Versus Pathological Cardiac Hypertrophy

P2860

Q24317248-286A0AB8-7A6C-4AD2-9D4C-A7CE84C7EACE Q26765624-2697791F-9127-41F5-BFD1-EF424156EE57 Q26796228-7F933BC6-A287-4090-948D-4CE30DA8E579 Q26994995-B419FE42-B1CA-4801-B54F-804686CC65E3 Q27016095-43D8B7C8-FBB4-42F1-B3AA-0C1F97540742 Q28277846-5E6D15E3-2718-4A62-B0AB-11626011A8CA Q28387081-BC7E179E-1037-4CBA-871E-DD0F389136B2 Q28397537-7FB98594-363E-40E2-9E28-78BB660765CC Q28487365-CA0E1A3F-02A0-4082-98EB-80FA8EC70841 Q28566041-2D7EEA50-0092-44BC-ABD6-12CB8771B40F Q28577899-0347E70C-3F89-4548-B7BD-83486D6CFEBC Q28592273-CFF1AEFC-5D4D-4F7D-9A04-DA1964B1E2A4 Q30377910-F45AEFD3-D542-404C-A226-08F041FC4402 Q33678033-4F7AEC90-77E2-459B-9BC8-6036BDF4E5B5 Q33715025-53C5D0AD-CDDD-4528-B2FD-C9497BA1EDF1 Q33792524-56B05EAF-BD39-436F-8174-B3D5AFFEC844 Q33832036-98F77912-3229-4FE2-BB30-6F31209BCFC8 Q33925323-F92DB494-6314-46B6-9210-1AECF550CF1F Q34201221-41474FA8-18D2-4EE9-81C6-742F76DF53A6 Q35062534-BAF841F1-C8CF-47A2-901D-82B8FF696826 Q35083510-AB7B6DC7-3979-47DD-8C80-F8E2BADBDA8F Q35158082-8AFBD8CA-499E-46F0-8BAA-820A1CB1C851 Q35165245-76A96C37-7E82-4829-878F-CF28D5EACC8E Q35182995-3664A940-9E58-4383-BBC4-8C59654ACABB Q35527626-C6D298FA-22C9-4552-B6D8-1C618C3A9520 Q35673862-A5CDD7F2-07A4-4ECA-8A5D-5D9756E1C159 Q35724660-543C93CC-60D3-4867-B9E3-10739DB4334F Q35880015-8F0BA838-AAE4-4D3C-BDD6-9281DD8F1C05 Q36024414-81DE4948-7B52-4D20-B2C3-E3A77F8D1396 Q36061754-59ED1177-0159-4386-8194-032B23A527BB Q36096234-79C9023D-2D4E-4053-AF07-B038BF2FD5C6 Q36220344-D2FBFA46-D7FC-4AAF-A608-B6F74CA18C36 Q36378477-F4D2B9C7-BA73-43CA-910D-B8DAF0BA9CDF Q36605039-B1BC1B1C-0CCF-4D33-A26B-EA6A406E8B39 Q36630478-9D623165-821E-4CF9-98B6-1D67797616BE Q36634367-07ABD979-05D0-4835-920E-6D66130975E8 Q36649830-EF89438B-488B-4605-B228-12FAF0B8D414 Q36679863-5C3D3B48-251C-40A4-A6DD-3EFFD8DF71ED Q36702291-069696E5-5031-4E1E-8921-21819B598B12 Q36780942-29D6E051-1FA6-4959-9ED2-FD4D50C1526A

P2860

Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q35578778

description

2007 nî lūn-bûn

@nan

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

2007年论文

@zh

2007年论文

@zh-cn

name

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@ast

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@en

type

label

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@ast

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@en

prefLabel

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@ast

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@en

P1433

Q35578778-B764EC60-B06B-478A-BEC0-1E204516AE6B

P1476

Q35578778-CA3AF975-A261-4285-8608-643623D67FC8

P2093

Q35578778-1C5AE540-8091-4A5C-A64F-899D6B06CD07

Q35578778-621A1473-A97D-493D-8B70-F634DAFDF878

Q35578778-69225EAE-5E08-4167-8316-D86F9ED00614

Q35578778-6ADF73D9-6DFD-4A0E-A309-512D2A2BEBCD

Q35578778-73D4DDD1-8B26-480A-B079-685103776407

Q35578778-8A265AEC-D28A-4782-9111-A170E0181B3E

Q35578778-8AAC4D4D-08C2-4277-9369-B1C8DC782858

Q35578778-B860B657-6A2C-4555-8E89-96E0A5DB9AC7

Q35578778-C0DE4D39-5C1B-4374-91A9-7375C8A34779

Q35578778-C9AC3037-AF72-4BB2-95C2-FC5A753CE5E8

P2860

Q35578778-02EA28DE-FF7D-4CF9-A0F3-C56887278CF0

Q35578778-043491AC-57F1-4A48-ADDF-87181B9C4908

Q35578778-34025CE9-98C7-4C9C-8C12-39A61268FBB7

Q35578778-347769ED-9776-4939-B6FE-7013B51AEC72

Q35578778-356AE389-198C-4460-81E8-C777C0C98B18

Q35578778-3EA5BC9A-D402-4389-B222-C578269443CD

Q35578778-43EAADB5-1578-423F-9C47-1B9A40225148

Q35578778-448FA488-F42D-4870-B8DF-6C7A1F8B7803

Q35578778-51541ABC-834B-48C0-99D5-3BBDD8B5687C

Q35578778-6B1C9D15-2E13-4962-9D4C-89D86BC2C114

P304

Q35578778-9EC4E63D-7358-49CE-9CD6-19ED0BFAF398

P31

Q35578778-2090BA88-BAA0-4F5D-BF78-4220C64B5FA9

P356

Q35578778-EC91D297-0576-45B9-8283-CA8DE4C160D6

P407

Q35578778-9EF6E168-D555-4FF3-8BD4-E53642877312

P433

Q35578778-5736CCB2-CCA3-48F4-B925-2D8A7F782765

P478

Q35578778-F7469811-CEB7-47EA-8EF4-7171D679317D

P50

Q35578778-41D0EF03-D2A2-42A7-A38A-CBC7DAB934C0

P577

Q35578778-3078D554-3842-4031-82DB-4812697711DB

P5875

Q35578778-E775194C-3322-44AA-BF57-A1562946BB64

P698

Q35578778-622E1FC7-6679-4618-8D28-61D3EA50BA6B

P932

Q35578778-B731E497-BA53-4E2C-B5AC-FE3567C6B082

P356

PNAS.0606663104

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Protective effects of exercise ...... d hypertrophic cardiomyopathy.

@en

P2093

Antje Buerger

http://www.w3.org/2001/XMLSchema#string

Elizabeth A Woodcock

http://www.w3.org/2001/XMLSchema#string

Fatemeh Amirahmadi

http://www.w3.org/2001/XMLSchema#string

Janelle P Mollica

http://www.w3.org/2001/XMLSchema#string

Julie R McMullen

http://www.w3.org/2001/XMLSchema#string

Kimberly A Hewitt

http://www.w3.org/2001/XMLSchema#string

Li Zhang

http://www.w3.org/2001/XMLSchema#string

Martina Schinke-Braun

http://www.w3.org/2001/XMLSchema#string

Patrick Y Jay

http://www.w3.org/2001/XMLSchema#string

Russell D Bouwman

http://www.w3.org/2001/XMLSchema#string

P2860

Myostatin regulates cardiomyocyte growth through modulation of Akt signaling

Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload

Critical role for the p110alpha phosphoinositide-3-OH kinase in growth and metabolic regulation

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

Health benefits of physical activity: the evidence.

Intermittent pressure overload triggers hypertrophy-independent cardiac dysfunction and vascular rarefaction.

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

Class IA phosphoinositide 3-kinase regulates heart size and physiological cardiac hypertrophy.

Akt/protein kinase B promotes organ growth in transgenic mice.

Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death.

P304

612-617

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.0606663104

http://www.w3.org/2001/XMLSchema#string

P407

P433

2

http://www.w3.org/2001/XMLSchema#string

P478

104

http://www.w3.org/2001/XMLSchema#string

P50

Garry L Jennings

P577

2007-01-03T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

6599242

http://www.w3.org/2001/XMLSchema#string

P698

17202264

http://www.w3.org/2001/XMLSchema#string

P932

1766433

http://www.w3.org/2001/XMLSchema#string