Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death.
about
Activation of protein synthesis in cardiomyocytes by the hypertrophic agent phenylephrine requires the activation of ERK and involves phosphorylation of tuberous sclerosis complex 2 (TSC2)Extracellular signal-regulated kinase 2 interacts with and is negatively regulated by the LIM-only protein FHL2 in cardiomyocytesPKC translocation and ERK1/2 activation in compensated right ventricular hypertrophy secondary to chronic emphysemaGDF-15 as a Target and Biomarker for Diabetes and Cardiovascular Diseases: A Translational ProspectiveExtracellular signal-regulated kinases 1/2 as regulators of cardiac hypertrophySecretogranin II; a protein increased in the myocardium and circulation in heart failure with cardioprotective propertiesThe alpha(1A/C)- and alpha(1B)-adrenergic receptors are required for physiological cardiac hypertrophy in the double-knockout mouseDUSP6 (MKP3) null mice show enhanced ERK1/2 phosphorylation at baseline and increased myocyte proliferation in the heart affecting disease susceptibilityRegulation of cardiac hypertrophic signaling by prolyl isomerase Pin1Protection of cardiomyocytes from ischemic/hypoxic cell death via Drbp1 and pMe2GlyDH in cardio-specific ARC transgenic miceZebrafish heart failure models for the evaluation of chemical probes and drugsEvidence for Altered Ca(2+) Handling in Growth Associated Protein 43-Knockout Skeletal MuscleDevelopmental stage-dependent effects of cardiac fibroblasts on function of stem cell-derived engineered cardiac tissues.Active kinase proteome screening reveals novel signal complexity in cardiomyopathy.The heart is an early target of anthrax lethal toxin in mice: a protective role for neuronal nitric oxide synthase (nNOS).Adult murine cardiomyocytes exhibit regenerative activity with cell cycle reentry through STAT3 in the healing process of myocarditis.Multikinase inhibitor sorafenib prevents pressure overload-induced left ventricular hypertrophy in rats by blocking the c-Raf/ERK1/2 signaling pathway.Direct and indirect interactions between calcineurin-NFAT and MEK1-extracellular signal-regulated kinase 1/2 signaling pathways regulate cardiac gene expression and cellular growth.Targeted deletion of ERK2 in cardiomyocytes attenuates hypertrophic response but provokes pathological stress induced cardiac dysfunction.Signaling responses after exposure to 5 alpha-dihydrotestosterone or 17 beta-estradiol in norepinephrine-induced hypertrophy of neonatal rat ventricular myocytesKMUP-1 attenuates isoprenaline-induced cardiac hypertrophy in rats through NO/cGMP/PKG and ERK1/2/calcineurin A pathwaysML290 is a biased allosteric agonist at the relaxin receptor RXFP1.Molecular determinants of the cardiometabolic phenotype.ERK-MAPK signaling opposes rho-kinase to reduce cardiomyocyte apoptosis in heart ischemic preconditioning.Differential activation of stress-response signaling in load-induced cardiac hypertrophy and failure.Regulator of G protein signaling 5 protects against cardiac hypertrophy and fibrosis during biomechanical stress of pressure overloadLeft ventricular global transcriptional profiling in human end-stage dilated cardiomyopathy.A novel chimeric natriuretic peptide reduces cardiomyocyte hypertrophy through the NHE-1-calcineurin pathway.Distinct cardiac transcriptional profiles defining pregnancy and exerciseExtracellular signal-regulated kinases 1 and 2 regulate the balance between eccentric and concentric cardiac growthMEK-ERK pathway modulation ameliorates disease phenotypes in a mouse model of Noonan syndrome associated with the Raf1(L613V) mutationCardiac titin and heart disease.Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation.Mechanical stress triggers cardiomyocyte autophagy through angiotensin II type 1 receptor-mediated p38MAP kinase independently of angiotensin II.Lactosylceramide promotes hypertrophy through ROS generation and activation of ERK1/2 in cardiomyocytes.Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy.Inhibition of signal transducer and activator of transcription 3 (STAT3) attenuates interleukin-6 (IL-6)-induced collagen synthesis and resultant hypertrophy in rat heart.Identification of Region-Specific Myocardial Gene Expression Patterns in a Chronic Swine Model of Repaired Tetralogy of Fallot.N-Acetyl Cysteine Inhibits Endothelin-1-Induced ROS Dependent Cardiac Hypertrophy through Superoxide Dismutase RegulationAngiotensin-(1-7) abrogates mitogen-stimulated proliferation of cardiac fibroblasts.
P2860
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P2860
Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death.
description
2002 nî lūn-bûn
@nan
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Involvement of extracellular s ...... ac hypertrophy and cell death.
@ast
Involvement of extracellular s ...... ac hypertrophy and cell death.
@en
Involvement of extracellular s ...... ac hypertrophy and cell death.
@nl
type
label
Involvement of extracellular s ...... ac hypertrophy and cell death.
@ast
Involvement of extracellular s ...... ac hypertrophy and cell death.
@en
Involvement of extracellular s ...... ac hypertrophy and cell death.
@nl
prefLabel
Involvement of extracellular s ...... ac hypertrophy and cell death.
@ast
Involvement of extracellular s ...... ac hypertrophy and cell death.
@en
Involvement of extracellular s ...... ac hypertrophy and cell death.
@nl
P1433
P1476
Involvement of extracellular s ...... ac hypertrophy and cell death.
@en
P2093
Orlando F Bueno
P304
P356
10.1161/01.RES.0000038488.38975.1A
P577
2002-11-01T00:00:00Z