Pathology of presynaptic proteins in Alzheimer's disease: more than simple loss of terminals.
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Alzheimer-related decrease in CYFIP2 links amyloid production to tau hyperphosphorylation and memory loss.MTM-6, a phosphoinositide phosphatase, is required to promote synapse formation in Caenorhabditis elegansOverexpression of 14-3-3z promotes tau phosphorylation at Ser262 and accelerates proteosomal degradation of synaptophysin in rat primary hippocampal neuronsBMS-708163 and Nilotinib restore synaptic dysfunction in human embryonic stem cell-derived Alzheimer's disease modelsTau-mediated synaptic damage in Alzheimer's diseaseSchizophrenia genes, gene expression, and neuropathology: on the matter of their convergenceApolipoprotein E4 domain interaction: synaptic and cognitive deficits in miceAPP transgenic modeling of Alzheimer's disease: mechanisms of neurodegeneration and aberrant neurogenesisPerforant path synaptic loss correlates with cognitive impairment and Alzheimer's disease in the oldest-oldSNAP-25 is a promising novel cerebrospinal fluid biomarker for synapse degeneration in Alzheimer's disease.Synapse loss in dementias.Roles of cholesterol and lipids in the etiopathogenesis of Alzheimer's disease.Synaptic change in the posterior cingulate gyrus in the progression of Alzheimer's diseaseRestored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer's diseaseCalpain dysregulation in Alzheimer's diseaseSynaptic deficits are rescued in the p25/Cdk5 model of neurodegeneration by the reduction of β-secretase (BACE1)Synaptic protein deficits are associated with dementia irrespective of extreme old ageTemporal dependence of cysteine protease activation following excitotoxic hippocampal injury.Loss of Munc18-1 long splice variant in GABAergic terminals is associated with cognitive decline and increased risk of dementia in a community sampleA combination extract of ginseng, epimedium, polygala, and tuber curcumae increases synaptophysin expression in APPV717I transgenic miceAnalysis of Proteins That Rapidly Change Upon Mechanistic/Mammalian Target of Rapamycin Complex 1 (mTORC1) Repression Identifies Parkinson Protein 7 (PARK7) as a Novel Protein Aberrantly Expressed in Tuberous Sclerosis Complex (TSC)Synaptic dysfunction and oxidative stress in Alzheimer's disease: emerging mechanisms.Aromatase Expression in the Hippocampus of AD Patients and 5xFAD Mice.Molecular mechanisms of the conjugated alpha,beta-unsaturated carbonyl derivatives: relevance to neurotoxicity and neurodegenerative diseases.RanBP9 overexpression accelerates loss of pre and postsynaptic proteins in the APΔE9 transgenic mouse brain.Impaired NGF/TrkA Signaling Causes Early AD-Linked Presynaptic Dysfunction in Cholinergic Primary NeuronsDecreased cortical FADD protein is associated with clinical dementia and cognitive decline in an elderly community sample.Effect of amyloids on the vesicular machinery: implications for somatic neurotransmission.Mitochondrial Dysfunction and Synaptic Transmission Failure in Alzheimer's Disease.Synaptic changes in frontotemporal lobar degeneration: correlation with MAPT haplotype and APOE genotype.Truncated tau deregulates synaptic markers in rat model for human tauopathy.Beta-amyloid disrupted synaptic vesicle endocytosis in cultured hippocampal neurons.Synaptophysin depletion and intraneuronal Aβ in organotypic hippocampal slice cultures from huAPP transgenic mice.Targeting synaptic pathology with a novel affinity mass spectrometry approach.Beta-amyloid-induced dynamin 1 depletion in hippocampal neurons. A potential mechanism for early cognitive decline in Alzheimer disease.Extracellular truncated tau causes early presynaptic dysfunction associated with Alzheimer's disease and other tauopathiesAbnormalities of presynaptic protein CDCrel-1 in striatum of rats reared in social isolation: relevance to neural connectivity in schizophrenia.Abnormal dendritic calcium activity and synaptic depotentiation occur early in a mouse model of Alzheimer's disease.Prenatal ethanol exposure in rats decreases levels of complexin proteins in the frontal cortex.Beneficial Effects of Exercise Pretreatment in a Sporadic Alzheimer's Rat Model.
P2860
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P2860
Pathology of presynaptic proteins in Alzheimer's disease: more than simple loss of terminals.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Pathology of presynaptic prote ...... than simple loss of terminals.
@ast
Pathology of presynaptic prote ...... than simple loss of terminals.
@en
type
label
Pathology of presynaptic prote ...... than simple loss of terminals.
@ast
Pathology of presynaptic prote ...... than simple loss of terminals.
@en
prefLabel
Pathology of presynaptic prote ...... than simple loss of terminals.
@ast
Pathology of presynaptic prote ...... than simple loss of terminals.
@en
P1476
Pathology of presynaptic prote ...... than simple loss of terminals
@en
P2093
William G Honer
P304
P356
10.1016/J.NEUROBIOLAGING.2003.04.005
P577
2003-12-01T00:00:00Z