XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
about
eIF5A regulation in response to inhibition of the nuclear export systemFirst-in-Class, First-in-Human Phase I Study of Selinexor, a Selective Inhibitor of Nuclear Export, in Patients With Advanced Solid TumorsTreatment of acquired drug resistance in multiple myeloma by combination therapy with XPO1 and topoisomerase II inhibitorsSelinexor (KPT-330) demonstrates anti-tumor efficacy in preclinical models of triple-negative breast cancer.Alternative Start Codon Connects eIF5A to Mitochondria.Insulin-like growth factor 2 mRNA-binding protein 1 (IGF2BP1) in cancer.XPO1 inhibition by selinexor induces potent cytotoxicity against high grade bladder malignancies
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P2860
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@ast
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@en
type
label
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@ast
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@en
prefLabel
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@ast
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@en
P2093
P2860
P1476
XPO1/CRM1 Inhibition Causes Antitumor Effects by Mitochondrial Accumulation of eIF5A.
@en
P2093
Anil K Sood
Behrouz Zand
David H Hawke
Dilara McCauley
Heather J Dalton
John E Wiktorowicz
Kshipra M Gharpure
Min Soon Cho
Morgan L Taylor
Rebecca A Previs
P2860
P304
P356
10.1158/1078-0432.CCR-14-1953
P407
P577
2015-04-15T00:00:00Z