Lipopolysaccharide binding protein enhances the responsiveness of alveolar macrophages to bacterial lipopolysaccharide. Implications for cytokine production in normal and injured lungs
about
Lipopolysaccharide is in close proximity to each of the proteins in its membrane receptor complex. transfer from CD14 to TLR4 and MD-2Balancing Innate Immunity and Inflammatory State via Modulation of Neutrophil Function: A Novel Strategy to Fight SepsisProduction of the acute-phase protein lipopolysaccharide-binding protein by respiratory type II epithelial cells: implications for local defense to bacterial endotoxinsHuman lipopolysaccharide-binding protein (LBP) and CD14 independently deliver triacylated lipoproteins to Toll-like receptor 1 (TLR1) and TLR2 and enhance formation of the ternary signaling complexInhibition of DNA synthesis in cultured hepatocytes by endotoxin-conditioned medium of activated stellate cells is transforming growth factor-beta and nitric oxide-independentICAM-1 and LFA-1 play critical roles in LPS-induced neutrophil recruitment into the alveolar space.Proteomic and computational analysis of bronchoalveolar proteins during the course of the acute respiratory distress syndromeLactoferrin inhibits the endotoxin interaction with CD14 by competition with the lipopolysaccharide-binding protein.Cell activation mediated by glycosylphosphatidylinositol-anchored or transmembrane forms of CD14.Lipopolysaccharide (LPS) recognition in macrophages. Participation of LPS-binding protein and CD14 in LPS-induced adaptation in rabbit peritoneal exudate macrophagesRole of plasma, lipopolysaccharide-binding protein, and CD14 in response of mouse peritoneal exudate macrophages to endotoxinAgmatine protects against zymosan-induced acute lung injury in mice by inhibiting NF-κB-mediated inflammatory response.A role for Toll-like receptor mediated signals in neutrophils in the pathogenesis of the anti-phospholipid syndrome.Role of house-dust endotoxin exposure in aetiology of allergy and asthma.Animal models of acute lung injuryPARP-1 inhibitor, DPQ, attenuates LPS-induced acute lung injury through inhibiting NF-κB-mediated inflammatory response.Roles for tumor necrosis factor alpha and nitric oxide in resistance of rat alveolar macrophages to Legionella pneumophila.Influence of lipopolysaccharide-binding protein on pulmonary inflammation in gram-negative pneumoniaModulating LPS signal transduction at the LPS receptor complex with synthetic Lipid A analoguesIntestinal Dysbiosis and Lowered Serum Lipopolysaccharide-Binding Protein in Parkinson's Disease.Innate immunity in the lungsLipopolysaccharide binding protein promoter variants influence the risk for Gram-negative bacteremia and mortality after allogeneic hematopoietic cell transplantation.Cell-free pool of CD14 mediates activation of transcription factor NF-kappa B by lipopolysaccharide in human endothelial cellsBacterial modulins: a novel class of virulence factors which cause host tissue pathology by inducing cytokine synthesisGlycyrrhizic Acid Prevents Sepsis-Induced Acute Lung Injury and Mortality in Rats.Apolipoprotein A-II augments monocyte responses to LPS by suppressing the inhibitory activity of LPS-binding protein.LPS-treated macrophage cytokines repress surfactant protein-B in lung epithelial cells.Endotoxin and cancer.Endotoxin and cancer.Sensing of microbial molecular patterns by Toll-like receptors.Assessment of immunotoxicity using precision-cut tissue slices.The role of lipopolysaccharide-binding protein in innate immunity: a revisit and its relevance to oral/periodontal health.Post-Effect of Air Quality Improvement on Biomarkers for Systemic Inflammation and Microparticles in Asthma Patients After the 2008 Beijing Olympic Games: a Pilot Study.Influence of synthetic antiendotoxin peptides on lipopolysaccharide (LPS) recognition and LPS-induced proinflammatory cytokine responses by cells expressing membrane-bound CD14CD14 and tissue factor expression by bacterial lipopolysaccharide-stimulated bovine alveolar macrophages in vitro.Human lipopolysaccharide-binding protein potentiates bactericidal activity of human bactericidal/permeability-increasing protein.Endotoxin activates human vascular smooth muscle cells despite lack of expression of CD14 mRNA or endogenous membrane CD14.Lipopolysaccharide binding protein is an essential component of the innate immune response to Escherichia coli peritonitis in mice.Evaluation of inflammatory and immune responses in long-term cultured human precision-cut lung slices.Respiratory epithelial cells regulate lung inflammation in response to inhaled endotoxin.
P2860
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P2860
Lipopolysaccharide binding protein enhances the responsiveness of alveolar macrophages to bacterial lipopolysaccharide. Implications for cytokine production in normal and injured lungs
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@ast
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@en
type
label
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@ast
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@en
prefLabel
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@ast
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@en
P2093
P2860
P356
P1476
Lipopolysaccharide binding pro ...... on in normal and injured lungs
@en
P2093
A M Moriarty
D J Letúrcq
J C Mathison
P S Tobias
R J Maunder
R J Ulevitch
T R Martin
P2860
P304
P356
10.1172/JCI116106
P407
P577
1992-12-01T00:00:00Z