Lipopolysaccharide binding protein is an essential component of the innate immune response to Escherichia coli peritonitis in mice.
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Balancing Innate Immunity and Inflammatory State via Modulation of Neutrophil Function: A Novel Strategy to Fight SepsisHepatocytes: a key cell type for innate immunityHuman lipopolysaccharide-binding protein (LBP) and CD14 independently deliver triacylated lipoproteins to Toll-like receptor 1 (TLR1) and TLR2 and enhance formation of the ternary signaling complexInfluence of lipopolysaccharide-binding protein on pulmonary inflammation in gram-negative pneumoniaApolipoprotein A-II augments monocyte responses to LPS by suppressing the inhibitory activity of LPS-binding protein.Trappin-2 promotes early clearance of Pseudomonas aeruginosa through CD14-dependent macrophage activation and neutrophil recruitmentTriggering receptor expressed on myeloid cells-2 fine-tunes inflammatory responses in murine Gram-negative sepsis.Distinct effects of Lactobacillus plantarum KL30B and Escherichia coli 3A1 on the induction and development of acute and chronic inflammation.CD44 deficiency is associated with enhanced Escherichia coli-induced proinflammatory cytokine and chemokine release by peritoneal macrophages.Acclimation capacity of the three-spined stickleback (Gasterosteus aculeatus, L.) to a sudden biological stress following a polymetallic exposure.A peritonitis model with low mortality and persisting intra-abdominal abscesses.
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Lipopolysaccharide binding protein is an essential component of the innate immune response to Escherichia coli peritonitis in mice.
description
2003 nî lūn-bûn
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2003年の論文
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2003年論文
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2003年論文
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name
Lipopolysaccharide binding pro ...... chia coli peritonitis in mice.
@en
type
label
Lipopolysaccharide binding pro ...... chia coli peritonitis in mice.
@en
prefLabel
Lipopolysaccharide binding pro ...... chia coli peritonitis in mice.
@en
P2860
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P1476
Lipopolysaccharide binding pro ...... chia coli peritonitis in mice.
@en
P2093
Alex F de Vos
Douglas T Golenbock
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10.1128/IAI.71.12.6747-6753.2003
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2003-12-01T00:00:00Z