Stemness of the hybrid Epithelial/Mesenchymal State in Breast Cancer and Its Association with Poor Survival.
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Phenotypic plasticity in prostate cancer: role of intrinsically disordered proteinsTumor Budding: The Name is EMT. Partial EMTImplications of the Hybrid Epithelial/Mesenchymal Phenotype in MetastasisIntratumoral Morphological Heterogeneity of Breast Cancer As an Indicator of the Metastatic Potential and Tumor Chemosensitivity.Inflammatory breast cancer: a model for investigating cluster-based dissemination.Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?EMT and MET: necessary or permissive for metastasis?Stability of the hybrid epithelial/mesenchymal phenotype.Jagged mediates differences in normal and tumor angiogenesis by affecting tip-stalk fate decision.Prolonged pemetrexed pretreatment augments persistence of cisplatin-induced DNA damage and eliminates resistant lung cancer stem-like cells associated with EMTEffect of Melatonin in Epithelial Mesenchymal Transition Markers and Invasive Properties of Breast Cancer Stem Cells of Canine and Human Cell LinesModeling the Transitions between Collective and Solitary Migration Phenotypes in Cancer Metastasis.Interleukin-like EMT inducer regulates partial phenotype switching in MITF-low melanoma cell linesClinically relevant morphological structures in breast cancer represent transcriptionally distinct tumor cell populations with varied degrees of epithelial-mesenchymal transition and CD44+CD24- stemness.Coupling the modules of EMT and stemness: A tunable 'stemness window' modelNotch-Jagged signalling can give rise to clusters of cells exhibiting a hybrid epithelial/mesenchymal phenotype.Suppression of the grainyhead transcription factor 2 gene (GRHL2) inhibits the proliferation, migration, invasion and mediates cell cycle arrest of ovarian cancer cells.Positive Quantitative Relationship between EMT and Contact-Initiated Sliding on Fiber-like Tracks.The GRHL2/ZEB Feedback Loop-A Key Axis in the Regulation of EMT in Breast Cancer.Evaluation of the correlation of vasculogenic mimicry, ALDH1, KiSS-1, and MACC1 in the prediction of metastasis and prognosis in ovarian carcinoma.Cell lineage determinants as regulators of breast cancer metastasis.Targeting the Epithelial-to-Mesenchymal Transition: The Case for Differentiation-Based Therapy.EMT and stemness: flexible processes tuned by alternative splicing in development and cancer progression.Emerging Biological Principles of Metastasis.Phenotypic Plasticity and Cell Fate Decisions in Cancer: Insights from Dynamical Systems TheoryEpithelial-mesenchymal transition, a spectrum of states: Role in lung development, homeostasis, and disease.Estimating real cell size distribution from cross-section microscopy imaging.Hybrid clone cells derived from human breast epithelial cells and human breast cancer cells exhibit properties of cancer stem/initiating cells.Complex Determinants of Epithelial: Mesenchymal Phenotypic Plasticity in Ovarian Cancer.Epithelial plasticity and cancer stem cells: Major mechanisms of cancer pathogenesis and therapy resistanceExpression and clinical relevance of epithelial and mesenchymal markers in circulating tumor cells from colorectal cancer.Quantifying the landscape and kinetic paths for epithelial-mesenchymal transition from a core circuit.Individualized Breast Cancer Characterization through Single-Cell Analysis of Tumor and Adjacent Normal Cells.Numb prevents a complete epithelial-mesenchymal transition by modulating Notch signalling.Predictive value of epithelial-mesenchymal-transition (EMT) signature and PARP-1 in prostate cancer radioresistance.Survival Outcomes in Cancer Patients Predicted by a Partial EMT Gene Expression Scoring Metric.The Ever-Evolving Concept of the Cancer Stem Cell in Pancreatic Cancer.Controlling Stochasticity in Epithelial-Mesenchymal Transition Through Multiple Intermediate Cellular States.Phenotypic Plasticity, Bet-Hedging, and Androgen Independence in Prostate Cancer: Role of Non-Genetic Heterogeneity.Disorder, Promiscuous Interactions, and Stochasticity Regulate State Switching in the Unstable Prostate.
P2860
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P2860
Stemness of the hybrid Epithelial/Mesenchymal State in Breast Cancer and Its Association with Poor Survival.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@ast
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@en
type
label
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@ast
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@en
prefLabel
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@ast
Stemness of the hybrid Epithel ...... ssociation with Poor Survival.
@en
P2093
P2860
P50
P1433
P1476
Stemness of the hybrid Epithel ...... Association with Poor Survival
@en
P2093
Anne Grosse-Wilde
Aymeric Fouquier d'Hérouël
Ellie McIntosh
Rolf E Kuestner
P2860
P304
P356
10.1371/JOURNAL.PONE.0126522
P407
P577
2015-05-28T00:00:00Z