HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications - Wikidata - awgldk - TriplyDB

HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

http://www.wikidata.org/entity/Q35657898

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Closed-loop optogenetic control of thalamus as a tool for interrupting seizures after cortical injury The Role of HCN Channels on Membrane Excitability in the Nervous System HCN4 subunit expression in fast-spiking interneurons of the rat spinal cord and hippocampus.Dysfunctional HCN ion channels in neurological diseases.Structural mechanism for the regulation of HCN ion channels by the accessory protein TRIP8b.Gabapentin Modulates HCN4 Channel Voltage-Dependence.Integrated Genome-wide association and hypothalamus eQTL studies indicate a link between the circadian rhythm-related gene PER1 and coping behavior.Everybody's Got the Fever…Be it Fahrenheit or Centigrade.Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies.Gain-of-function HCN2 variants in genetic epilepsy.Disturbed Processing of Contextual Information in HCN3 Channel Deficient Mice.Ih Interacts with Somato-Dendritic Structure to Determine Frequency Response to Weak Alternating Electric Field Stimulation.Developmental Changes in HCN Channel Modulation of Neocortical Layer 1 Interneurons.Dynamical Mechanism of Hyperpolarization-Activated Non-specific Cation Current Induced Resonance and Spike-Timing Precision in a Neuronal Model.Functional variants in HCN4 and CACNA1H may contribute to genetic generalized epilepsy.Preclinical Comparison of Mechanistically Different Antiseizure, Antinociceptive, and/or Antidepressant Drugs in a Battery of Rodent Models of Nociceptive and Neuropathic Pain.A Loss-of-Function Mutation Associated With Familial Benign Myoclonic Epilepsy in Infancy Causes Increased Neuronal Excitability

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

http://www.wikidata.org/entity/Q35657898

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

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J.1476-5381.2011.01507.X

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British Journal of Pharmacology

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HCN channelopathies: pathophysiology in genetic epilepsy and therapeutic implications

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A Marie Phillips

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Christopher A Reid

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Steven Petrou

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HCN1 channel subunits are a molecular substrate for hypnotic actions of ketamine.

Enhanced expression of a specific hyperpolarization-activated cyclic nucleotide-gated cation channel (HCN) in surviving dentate gyrus granule cells of human and experimental epileptic hippocampus

Revised terminology and concepts for organization of seizures and epilepsies: report of the ILAE Commission on Classification and Terminology, 2005-2009

Seizures and enhanced cortical GABAergic inhibition in two mouse models of human autosomal dominant nocturnal frontal lobe epilepsy.

Environmental manipulations early in development alter seizure activity, Ih and HCN1 protein expression later in life

Rab8b and its interacting partner TRIP8b are involved in regulated secretion in AtT20 cells

Association with the auxiliary subunit PEX5R/Trip8b controls responsiveness of HCN channels to cAMP and adrenergic stimulation

Regulation of HCN channel surface expression by a novel C-terminal protein-protein interaction

Nav1.1 localizes to axons of parvalbumin-positive inhibitory interneurons: a circuit basis for epileptic seizures in mice carrying an Scn1a gene mutation

Subunit-specific effects of isoflurane on neuronal Ih in HCN1 knockout mice.

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10.1111/J.1476-5381.2011.01507.X

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