Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy - Wikidata - awgldk - TriplyDB

Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy

Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy

http://www.wikidata.org/entity/Q35659099

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Ameliorative effect of fisetin on cisplatin-induced nephrotoxicity in rats via modulation of NF-κB activation and antioxidant defence Mitochondria-Targeted Antioxidants: Future Perspectives in Kidney Ischemia Reperfusion Injury Role of endoplasmic reticulum stress in drug-induced toxicity Mitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modifications Targeting Mitochondria and Reactive Oxygen Species-Driven Pathogenesis in Diabetic Nephropathy MitoQ blunts mitochondrial and renal damage during cold preservation of porcine kidneys Cisplatin induces a mitochondrial-ROS response that contributes to cytotoxicity depending on mitochondrial redox status and bioenergetic functions Mitochondrial modulation by Epigallocatechin 3-Gallate ameliorates cisplatin induced renal injury through decreasing oxidative/nitrative stress, inflammation and NF-kB in mice Baicalein, a Bioflavonoid, Prevents Cisplatin-Induced Acute Kidney Injury by Up-Regulating Antioxidant Defenses and Down-Regulating the MAPKs and NF-κB Pathways Cellular and Molecular Mechanisms of AKI.Tempol, an intracellular antioxidant, inhibits tissue factor expression, attenuates dendritic cell function, and is partially protective in a murine model of cerebral malaria.Effect of mitochondrially targeted carboxy proxyl nitroxide on Akt-mediated survival in Daudi cells: Significance of a dual mode of action Mitochondrial dysfunction in inherited renal disease and acute kidney injury PGC-1α attenuates hydrogen peroxide-induced apoptotic cell death by upregulating Nrf-2 via GSK3β inactivation mediated by activated p38 in HK-2 Cells.Late-onset Mitochondrial Cardiomyopathy Triggered by Anticancer Treatment Therapeutic targeting of the mitochondrial dysfunction in septic acute kidney injury.Pathophysiological mechanisms of catecholamine and cocaine-mediated cardiotoxicity.Computational modeling of apoptotic signaling pathways induced by cisplatin Mitochondrial dysregulation and protection in cisplatin nephrotoxicity.Mitochondria-targeted vitamin E analogs inhibit breast cancer cell energy metabolism and promote cell death.Cannabidiol Protects against Doxorubicin-Induced Cardiomyopathy by Modulating Mitochondrial Function and Biogenesis.Mechanism of Cisplatin-Induced Cytotoxicity Is Correlated to Impaired Metabolism Due to Mitochondrial ROS Generation.Neuroprotective Effects of Mitochondria-Targeted Plastoquinone and Thymoquinone in a Rat Model of Brain Ischemia/Reperfusion Injury.Antiproliferative effects of mitochondria-targeted cationic antioxidants and analogs: Role of mitochondrial bioenergetics and energy-sensing mechanism β-Caryophyllene ameliorates cisplatin-induced nephrotoxicity in a cannabinoid 2 receptor-dependent manner.Mitigation of NADPH Oxidase 2 Activity as a Strategy to Inhibit Peroxynitrite Formation.Mitochondrial reactive oxygen species generation triggers inflammatory response and tissue injury associated with hepatic ischemia-reperfusion: therapeutic potential of mitochondrially targeted antioxidants.Puerarin attenuates cisplatin-induced rat nephrotoxicity: The involvement of TLR4/NF-κB signaling pathway Cisplatin nephrotoxicity involves mitochondrial injury with impaired tubular mitochondrial enzyme activity.Angiotensin II type 1 receptor blockers increase tolerance of cells to copper and cisplatin.Drug-induced mitochondrial dysfunction and cardiotoxicity.The novel, orally available and peripherally restricted selective cannabinoid CB2 receptor agonist LEI-101 prevents cisplatin-induced nephrotoxicity.Mitochondria-targeted drugs synergize with 2-deoxyglucose to trigger breast cancer cell death.Novel orally active epoxyeicosatrienoic acid (EET) analogs attenuate cisplatin nephrotoxicity.Mitochondria-targeted heme oxygenase-1 decreases oxidative stress in renal epithelial cells Increasing cGMP-dependent protein kinase I activity attenuates cisplatin-induced kidney injury through protection of mitochondria function Protective effect of metalloporphyrins against cisplatin-induced kidney injury in mice.Epicatechin limits renal injury by mitochondrial protection in cisplatin nephropathy.Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?A review on the role of quinones in renal disorders.

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Mitochondrial-targeted antioxidants represent a promising approach for prevention of cisplatin-induced nephropathy

http://www.wikidata.org/entity/Q35659099

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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J.FREERADBIOMED.2011.11.001

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Free Radical Biology and Medicine

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Mitochondrial-targeted antioxi ...... cisplatin-induced nephropathy

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Balaraman Kalyanaraman

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Béla Horváth

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Eileen Holovac

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Galin Tanchian

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Isaac E Stillman

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Joy Joseph

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Malek Kechrid

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Samir M Parikh

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Vivek Patel

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P2860

Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury

Nitric oxide and peroxynitrite in health and disease

Peroxynitrite: biochemistry, pathophysiology and development of therapeutics

Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity

Cellular processing of platinum anticancer drugs

Regulation of mitochondrial dynamics in acute kidney injury in cell culture and rodent models.

Cannabinoid-2 receptor limits inflammation, oxidative/nitrosative stress, and cell death in nephropathy.

The antioxidant transcription factor Nrf2 negatively regulates autophagy and growth arrest induced by the anticancer redox agent mitoquinone

Clonogenic assay of cells in vitro.

Nephrotoxicity induced by cancer chemotherapy with special emphasis on cisplatin toxicity.

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497-506

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10.1016/J.FREERADBIOMED.2011.11.001

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2

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Partha Mukhopadhyay

Zsuzsanna K Zsengeller

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2011-11-10T00:00:00Z

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51837236

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22120494

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3253235

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