Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism. - Wikidata - awgldk - TriplyDB

Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.

Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.

http://www.wikidata.org/entity/Q35685680

about

Neural mechanisms of pain and alcohol dependence Animal models of post-traumatic stress disorder and recent neurobiological insights.Neuropeptide Y (NPY) in the extended amygdala is recruited during the transition to alcohol dependence Anticipatory 50 kHz ultrasonic vocalizations are associated with escalated alcohol intake in dependent rats.Predator odor stress alters corticotropin-releasing factor-1 receptor (CRF1R)-dependent behaviors in rats.Nicotine dependence produces hyperalgesia: role of corticotropin-releasing factor-1 receptors (CRF1Rs) in the central amygdala (CeA).Corticotropin-releasing factor (CRF) and α 2 adrenergic receptors mediate heroin withdrawal-potentiated startle in rats Chronic CRF1 receptor blockade reduces heroin intake escalation and dependence-induced hyperalgesia Clarifying the roles of homeostasis and allostasis in physiological regulation.Structural reorganization of pyramidal neurons in the medial prefrontal cortex of alcohol dependent rats is associated with altered glial plasticity.Pain is associated with risky drinking over time among HIV-infected persons in St. Petersburg, Russia.Increased alcohol consumption in urocortin 3 knockout mice is unaffected by chronic inflammatory pain.Hypocretin receptor 2 antagonism dose-dependently reduces escalated heroin self-administration in rats.Protracted abstinence from chronic ethanol exposure alters the structure of neurons and expression of oligodendrocytes and myelin in the medial prefrontal cortex.Inflammatory Pain Promotes Increased Opioid Self-Administration: Role of Dysregulated Ventral Tegmental Area μ Opioid Receptors.Corticosteroid-dependent plasticity mediates compulsive alcohol drinking in rats.Divergent regulation of distinct glucocorticoid systems in alcohol dependence.The affective dimension of pain as a risk factor for drug and alcohol addiction.Chronic intermittent voluntary alcohol drinking induces hyperalgesia in Sprague-Dawley rats.Alcohol dependence as a chronic pain disorder The NK1 receptor antagonist L822429 reduces heroin reinforcement.11β-hydroxysteroid dehydrogenase inhibition as a new potential therapeutic target for alcohol abuse Long-term antagonism of κ opioid receptors prevents escalation of and increased motivation for heroin intake.Neuronal extracellular signal-regulated kinase (ERK) activity as marker and mediator of alcohol and opioid dependence.The Prefrontal Cortex as a Critical Gate of Negative Affect and Motivation in Alcohol Use Disorder.Involvement of delta opioid receptors in alcohol withdrawal-induced mechanical allodynia in male C57BL/6 mice.Pain and Risk Behaviors Among HIV-Infected Persons in St. Petersburg, Russia.Traumatic stress reactivity promotes excessive alcohol drinking and alters the balance of prefrontal cortex-amygdala activity Electroacupuncture Attenuates Hyperalgesia in Rats Withdrawn from Chronic Alcohol Drinking via Habenular Mu Opioid Receptors.Opiate dependence induces cell type-specific plasticity of intrinsic membrane properties in the rat juxtacapsular bed nucleus of stria terminalis (jcBNST).Withdrawal from Chronic Nicotine Exposure Produces Region-Specific Tolerance to Alcohol-Stimulated GluA1 Phosphorylation.Neurobiological Correlates of Pain Avoidance-Like Behavior in Morphine-Dependent and Non-Dependent Rats.Corticotropin-Releasing Factor (CRF) Neurocircuitry and Neuropharmacology in Alcohol Drinking.Compulsive-Like Sufentanil Vapor Self-Administration in Rats.Voluntary induction and maintenance of alcohol dependence in rats using alcohol vapor self-administration.Negative reinforcement via motivational withdrawal is the driving force behind the transition to addiction.Inhibition of Glyoxalase 1 reduces alcohol self-administration in dependent and nondependent rats.Systemic and Intra-Habenular Activation of the Orphan G Protein-Coupled Receptor GPR139 Decreases Compulsive-Like Alcohol Drinking and Hyperalgesia in Alcohol-Dependent Rats.An enzymatic approach reverses nicotine dependence, decreases compulsive-like intake, and prevents relapse

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Development of mechanical hypersensitivity in rats during heroin and ethanol dependence: alleviation by CRF₁ receptor antagonism.

http://www.wikidata.org/entity/Q35685680

description

2011 nî lūn-bûn

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2011年の論文

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2011年论文

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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J.NEUROPHARM.2011.11.006

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Neuropharmacology

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Development of mechanical hype ...... n by CRF₁ receptor antagonism.

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George F Koob

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Gery Schulteis

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Joel E Schlosburg

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Kaushik K Misra

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Leandro F Vendruscolo

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Paula E Park

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Scott Edwards

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Sunmee Wee

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P2860

Opioids, pain, the brain, and hyperkatifeia: a framework for the rational use of opioids for pain

A role for brain stress systems in addiction.

Progressive enhancement of delayed hyperalgesia induced by repeated heroin administration: a sensitization process

Cellular and molecular mechanisms of drug dependence

Opioid-induced hyperalgesia: a qualitative systematic review

Pain-related anxiety-like behavior requires CRF1 receptors in the amygdala

Drug abuse: hedonic homeostatic dysregulation

Quantitative assessment of tactile allodynia in the rat paw

Role of corticotropin-releasing factor in drug addiction: potential for pharmacological intervention.

Corticotropin-releasing factor (CRF) sensitization of ethanol withdrawal-induced anxiety-like behavior is brain site specific and mediated by CRF-1 receptors: relation to stress-induced sensitization

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1142-1151

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10.1016/J.NEUROPHARM.2011.11.006

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2

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62

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2011-11-23T00:00:00Z

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51836799

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22119954

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3262951

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