RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma - Wikidata - awgldk - TriplyDB

RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

http://www.wikidata.org/entity/Q35690596

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Pancreatic Cancer Metabolism: Breaking It Down to Build It Back Up GEMMs as preclinical models for testing pancreatic cancer therapies Pancreatic cancer modeling using retrograde viral vector delivery and in vivo CRISPR/Cas9-mediated somatic genome editing.Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression.For robust big data analyses: a collection of 150 important pro-metastatic genes The Impact of SMAD4 Loss on Outcome in Patients with Advanced Pancreatic Cancer Treated with Systemic Chemotherapy An in vivo multiplexed small-molecule screening platform.Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization.Human phosphatase CDC14A is recruited to the cell leading edge to regulate cell migration and adhesion.Angiogenic gene signature in human pancreatic cancer correlates with TGF-beta and inflammatory transcriptomes.Improving the reliability of peer-reviewed publications: We are all in it together.RUNX3 defines disease behavior in pancreatic ductal adenocarcinoma.Characterization of RON protein isoforms in pancreatic cancer: implications for biology and therapeutics.Clinicopathological significance of SMAD4 loss in pancreatic ductal adenocarcinomas: a systematic review and meta-analysis.The role of hypoxia in pancreatic cancer: a potential therapeutic target?Enhancer Reprogramming Promotes Pancreatic Cancer Metastasis.Exosomes: potential for early detection in pancreatic cancer.The Emerging Roles of RUNX Transcription Factors in Epithelial-Mesenchymal Transition.Runx3 in Immunity, Inflammation and Cancer.Pancreatic cancer biology and genetics from an evolutionary perspective.Five-Year Actual Overall Survival in Resected Pancreatic Cancer: A Contemporary Single-Institution Experience from a Multidisciplinary Perspective.The Role of Consolidation Chemo-Radiotherapy in Locally Advanced Pancreatic Cancer Receiving Chemotherapy: An Updated Systematic Review and Meta-Analysis.UnPAKing RUNX3 functions-Both sides of the coin.Recurrent noncoding regulatory mutations in pancreatic ductal adenocarcinoma.Acinar-to-Ductal Metaplasia Induced by Transforming Growth Factor Beta Facilitates KRASG12D-driven Pancreatic Tumorigenesis.Epithelial-to-mesenchymal transition confers pericyte properties on cancer cells.Disconnect between EMT and metastasis in pancreas cancer Survival of pancreatic cancer cells lacking KRAS function.BLIMP1 Induces Transient Metastatic Heterogeneity in Pancreatic Cancer.Transcriptional regulation by RUNX3 Regulation of RUNX3 expression and activity RUNX3 Regulates Immune Response and Cell Migration RUNX3 as a Potential Predictor of Metastasis in Human Pancreatic Cancer.Runx3 plays a critical role in restriction-point and defense against cellular transformation.RUNX1 promote invasiveness in pancreatic ductal adenocarcinoma through regulating miR-93.PDAC-derived exosomes enrich the microenvironment in MDSCs in a SMAD4-dependent manner through a new calcium related axis.Combination immunohistochemistry for SMAD4 and Runt-related transcription factor 3 may identify a favorable prognostic subgroup of pancreatic ductal adenocarcinomas.Functional Analyses of RUNX3 and CaMKIINα in Ovarian Cancer Cell Lines Reveal Tumor-Suppressive Functions for CaMKIINα and Dichotomous Roles for RUNX3 Transcript Variants.The evolution into personalized therapies in pancreatic ductal adenocarcinoma: challenges and opportunities.Understanding Disease Biology and Informing the Management of Pancreas Cancer With Preclinical Model Systems.

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RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

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2015年论文

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RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

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RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

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RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

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RUNX3 Controls a Metastatic Switch in Pancreatic Ductal Adenocarcinoma

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Amy E Chang

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Kamel Izeradjene

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Kathleen E DelGiorno

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Libing Feng

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P2860

Preoperative/neoadjuvant therapy in pancreatic cancer: a systematic review and meta-analysis of response and resection percentages

Kras(G12D) and Smad4/Dpc4 haploinsufficiency cooperate to induce mucinous cystic neoplasms and invasive adenocarcinoma of the pancreas

Runx3 inactivation is a crucial early event in the development of lung adenocarcinoma

Genetic basis of pancreas cancer development and progression: insights from whole-exome and whole-genome sequencing

Cancer invasion and the microenvironment: plasticity and reciprocity

DPC4 gene status of the primary carcinoma correlates with patterns of failure in patients with pancreatic cancer.

The RUNX genes: gain or loss of function in cancer

Causal relationship between the loss of RUNX3 expression and gastric cancer

Pancreatic cancer

Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice

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