Selective subversion of autophagy complexes facilitates completion of the Brucella intracellular cycle. - Wikidata - awgldk - TriplyDB

Selective subversion of autophagy complexes facilitates completion of the Brucella intracellular cycle.

Selective subversion of autophagy complexes facilitates completion of the Brucella intracellular cycle.

http://www.wikidata.org/entity/Q35697369

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Regulation of ATG4B stability by RNF5 limits basal levels of autophagy and influences susceptibility to bacterial infection The Mucosal Immune System and Its Regulation by Autophagy Establishment of Chronic Infection: Brucella's Stealth Strategy Modulation of Host Autophagy during Bacterial Infection: Sabotaging Host Munitions for Pathogen Nutrition Interactions between Shigella flexneri and the Autophagy Machinery Type IV secretion system of Brucella spp. and its effectors PAMPs and DAMPs: signal 0s that spur autophagy and immunity Autophagy modulation as a potential therapeutic target for diverse diseases Autophagy in the regulation of pathogen replication and adaptive immunity Cross-talk between bile acids and intestinal microbiota in host metabolism and health Entrance and survival of Brucella pinnipedialis hooded seal strain in human macrophages and epithelial cells Contrasting Lifestyles Within the Host Cell Space: A Final Frontier for Vacuolar Pathogens Secretory autophagy Yip1A, a novel host factor for the activation of the IRE1 pathway of the unfolded protein response during Brucella infection Brucella induces an unfolded protein response via TcpB that supports intracellular replication in macrophages Selective autophagy: xenophagy Ehrlichia secretes Etf-1 to induce autophagy and capture nutrients for its growth through RAB5 and class III phosphatidylinositol 3-kinase Autophagosomes induced by a bacterial Beclin 1 binding protein facilitate obligatory intracellular infection The invA gene of Brucella melitensis is involved in intracellular invasion and is required to establish infection in a mouse model Brucella Dysregulates Monocytes and Inhibits Macrophage Polarization through LC3-Dependent Autophagy.Mechanism of Asp24 upregulation in Brucella abortus rough mutant with a disrupted O-antigen export system and effect of Asp24 in bacterial intracellular survival.The parasitophorous vacuole membrane of Toxoplasma gondii is targeted for disruption by ubiquitin-like conjugation systems of autophagy.Bactericidal Effect of Silver Nanoparticles on Intramacrophage Brucella abortus 544.Transcriptome-Wide Identification of Hfq-Associated RNAs in Brucella suis by Deep Sequencing.Bacterial colonization of host cells in the absence of cholesterol.The predicted ABC transporter AbcEDCBA is required for type IV secretion system expression and lysosomal evasion by Brucella ovis.Tripping on acid: trans-kingdom perspectives on biological acids in immunity and pathogenesis.Francisella tularensis harvests nutrients derived via ATG5-independent autophagy to support intracellular growth.Systems biology analysis of Brucella infected Peyer's patch reveals rapid invasion with modest transient perturbations of the host transcriptome.The autophagic machinery ensures nonlytic transmission of mycobacteria.Replication of Brucella abortus and Brucella melitensis in fibroblasts does not require Atg5-dependent macroautophagy.The functional and pathologic relevance of autophagy proteases.Autophagy and its effects: making sense of double-edged swords.Pathogenesis and immunobiology of brucellosis: review of Brucella-host interactions The role of autophagy in intracellular pathogen nutrient acquisition.The abcEDCBA-Encoded ABC Transporter and the virB Operon-Encoded Type IV Secretion System of Brucella ovis Are Critical for Intracellular Trafficking and Survival in Ovine Monocyte-Derived Macrophages.ATG16L1 deficiency in macrophages drives clearance of uropathogenic E. coli in an IL-1β-dependent manner Nondegradative role of Atg5-Atg12/ Atg16L1 autophagy protein complex in antiviral activity of interferon gamma.Two systems for targeted gene deletion in Coxiella burnetii

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P2860

Selective subversion of autophagy complexes facilitates completion of the Brucella intracellular cycle.

http://www.wikidata.org/entity/Q35697369

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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name

Selective subversion of autoph ...... Brucella intracellular cycle.

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Selective subversion of autoph ...... Brucella intracellular cycle.

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type

label

Selective subversion of autoph ...... Brucella intracellular cycle.

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Selective subversion of autoph ...... Brucella intracellular cycle.

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prefLabel

Selective subversion of autoph ...... Brucella intracellular cycle.

@ast

Selective subversion of autoph ...... Brucella intracellular cycle.

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J.CHOM.2011.12.002

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Cell Host & Microbe

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Selective subversion of autoph ...... Brucella intracellular cycle.

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P2093

Bryan Hansen

http://www.w3.org/2001/XMLSchema#string

Robert Child

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Tara D Wehrly

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Tregei Starr

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P2860

Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages

A phosphatidylinositol 3-kinase class III sub-complex containing VPS15, VPS34, Beclin 1, UVRAG and BIF-1 regulates cytokinesis and degradative endocytic traffic

Autophagy in immunity and inflammation

Distinct regulation of autophagic activity by Atg14L and Rubicon associated with Beclin 1-phosphatidylinositol-3-kinase complex

Autophagy is essential for mouse sense of balance

Methods in mammalian autophagy research

Regulation mechanisms and signaling pathways of autophagy

Autophagy is a defense mechanism inhibiting BCG and Mycobacterium tuberculosis survival in infected macrophages

Beclin 1 forms two distinct phosphatidylinositol 3-kinase complexes with mammalian Atg14 and UVRAG

Autophagy is activated for cell survival after endoplasmic reticulum stress

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33-45

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10.1016/J.CHOM.2011.12.002

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1

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11

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Herbert W. "Skip" Virgin

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221765862

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