Cortical deficits of glutamic acid decarboxylase 67 expression in schizophrenia: clinical, protein, and cell type-specific features.
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Cortical parvalbumin interneurons and cognitive dysfunction in schizophreniaSynchrony in schizophrenia: a window into circuit-level pathophysiologyHigh-frequency oscillations and the neurobiology of schizophreniaPrenatal stress and inhibitory neuron systems: implications for neuropsychiatric disordersGABA abnormalities in schizophrenia: a methodological review of in vivo studiesSchizophrenia as a disorder of molecular pathwaysTranscriptional regulation of GAD1 GABA synthesis gene in the prefrontal cortex of subjects with schizophreniaPrenatal choline and the development of schizophreniaThe role of glutamatergic inputs onto parvalbumin-positive interneurons: relevance for schizophreniaDevelopmental origins of brain disorders: roles for dopamineMaternal immune activation leads to selective functional deficits in offspring parvalbumin interneurons.Inhibition of parvalbumin-expressing interneurons results in complex behavioral changes.Different Paths to Core Pathology: The Equifinal Model of the Schizophrenia SyndromeMolecular substrates of schizophrenia: homeostatic signaling to connectivityPyramidal cell selective ablation of N-methyl-D-aspartate receptor 1 causes increase in cellular and network excitability.Inhibition of protein translation by the DISC1-Boymaw fusion gene from a Scottish family with major psychiatric disorders.The role of cannabinoid 1 receptor expressing interneurons in behavior.Reduced glutamate decarboxylase 65 protein within primary auditory cortex inhibitory boutons in schizophrenia.Prolonged Ketamine Effects in Sp4 Hypomorphic Mice: Mimicking Phenotypes of Schizophrenia.Myelination of parvalbumin interneurons: a parsimonious locus of pathophysiological convergence in schizophreniaProfiles of VGF Peptides in the Rat Brain and Their Modulations after Phencyclidine Treatment.Cortical inhibitory neuron disturbances in schizophrenia: role of the ontogenetic transcription factor Lhx6Stem cell therapy: a new approach to the treatment of refractory depression.Lower expression of glutamic acid decarboxylase 67 in the prefrontal cortex in schizophrenia: contribution of altered regulation by Zif268Epigenetic mechanisms in schizophrenia.Alterations in cortical network oscillations and parvalbumin neurons in schizophrenia.Pathological Basis for Deficient Excitatory Drive to Cortical Parvalbumin Interneurons in Schizophrenia.Lower glutamic acid decarboxylase 65-kDa isoform messenger RNA and protein levels in the prefrontal cortex in schizoaffective disorder but not schizophreniaAssociation study of OPRM1 polymorphisms with Schizophrenia in Han Chinese population.Neurodevelopment, GABA system dysfunction, and schizophreniaAltered cortical expression of GABA-related genes in schizophrenia: illness progression vs developmental disturbanceMolecular profiles of parvalbumin-immunoreactive neurons in the superior temporal cortex in schizophrenia.STEP levels are unchanged in pre-frontal cortex and associative striatum in post-mortem human brain samples from subjects with schizophrenia, bipolar disorder and major depressive disorderElevated Hippocampal Cholinergic Neurostimulating Peptide precursor protein (HCNP-pp) mRNA in the amygdala in major depression.Chemokine receptors and cortical interneuron dysfunction in schizophrenia.Altered expression of CDC42 signaling pathway components in cortical layer 3 pyramidal cells in schizophrenia.Transcriptional dysregulation of γ-aminobutyric acid transporter in parvalbumin-containing inhibitory neurons in the prefrontal cortex in schizophrenia.Dysregulated ErbB4 Splicing in Schizophrenia: Selective Effects on Parvalbumin ExpressionMaternal immune activation by LPS selectively alters specific gene expression profiles of interneuron migration and oxidative stress in the fetus without triggering a fetal immune responseEffects of chronic inhibition of GABA synthesis on attention and impulse control.
P2860
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P2860
Cortical deficits of glutamic acid decarboxylase 67 expression in schizophrenia: clinical, protein, and cell type-specific features.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Cortical deficits of glutamic ...... d cell type-specific features.
@ast
Cortical deficits of glutamic ...... d cell type-specific features.
@en
type
label
Cortical deficits of glutamic ...... d cell type-specific features.
@ast
Cortical deficits of glutamic ...... d cell type-specific features.
@en
prefLabel
Cortical deficits of glutamic ...... d cell type-specific features.
@ast
Cortical deficits of glutamic ...... d cell type-specific features.
@en
P2093
P2860
P1476
Cortical deficits of glutamic ...... d cell type-specific features.
@en
P2093
Allan R Sampson
Allison A Curley
David W Volk
Dominique Arion
Josephine K Asafu-Adjei
Kenneth N Fish
P2860
P304
P356
10.1176/APPI.AJP.2011.11010052
P407
P577
2011-06-01T00:00:00Z