ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
about
The potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophyThe TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.ADAM12 and alpha9beta1 integrin are instrumental in human myogenic cell differentiation.TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokineA human-specific deletion in mouse Cmah increases disease severity in the mdx model of Duchenne muscular dystrophyDistinct genetic regions modify specific muscle groups in muscular dystrophy.Therapeutic approaches to muscular dystrophy.Duchenne muscular dystrophy gene therapy: Lost in translation?Transgenic overexpression of dystroglycan does not inhibit muscular dystrophy in mdx miceTransgenic expression of {alpha}7{beta}1 integrin maintains muscle integrity, increases regenerative capacity, promotes hypertrophy, and reduces cardiomyopathy in dystrophic mice.Potential role for ADAM15 in pathological neovascularization in mice.Characterization of a novel chicken muscle disorder through differential gene expression and pathway analysis using RNA-sequencing.Overexpression of the cytotoxic T cell (CT) carbohydrate inhibits muscular dystrophy in the dyW mouse model of congenital muscular dystrophy 1A.Transgenic overexpression of ADAM12 suppresses muscle regeneration and aggravates dystrophy in aged mdx mice.A contemporary atlas of the mouse diaphragm: myogenicity, vascularity, and the Pax3 connection.ZNF555 protein binds to transcriptional activator site of 4qA allele and ANT1: potential implication in Facioscapulohumeral dystrophyIncreasing alpha 7 beta 1-integrin promotes muscle cell proliferation, adhesion, and resistance to apoptosis without changing gene expression.Overexpression of Galgt2 reduces dystrophic pathology in the skeletal muscles of alpha sarcoglycan-deficient miceADAM function in embryogenesisCell-matrix interactions in muscle disease.Comparison of satellite cell-derived myoblasts and C2C12 differentiation in two- and three-dimensional cultures: changes in adhesion protein expression.Role of metalloprotease disintegrin ADAM12 in determination of quiescent reserve cells during myogenic differentiation in vitro.Regulation of ADAM12 cell-surface expression by protein kinase C epsilon.Cooperation of the metalloprotease, disintegrin, and cysteine-rich domains of ADAM12 during inhibition of myogenic differentiation.Muscle genome-wide expression profiling during disease evolution in mdx mice.Identification of novel genes significantly affecting growth in catfish through GWAS analysis.Novel metalloprotease-disintegrin, meltrin epsilon (ADAM35), expressed in epithelial tissues during chick embryogenesis.ADAM12-S Stimulates Bone Growth in Transgenic Mice by Modulating Chondrocyte Proliferation and Maturation
P2860
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P2860
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@ast
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@en
type
label
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@ast
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@en
prefLabel
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@ast
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@en
P2093
P2860
P50
P1476
ADAM12 alleviates the skeletal muscle pathology in mdx dystrophic mice.
@en
P2093
Behzad Moghadaszadeh
Camilla Fröhlich
Eva Engvall
Nobuko Kawaguchi
Pauliina Kronqvist
Ulla M Wewer
Xiufeng Xu
P2860
P304
P356
10.1016/S0002-9440(10)64431-8
P407
P577
2002-11-01T00:00:00Z