Exercise reverses preamyloid oligomer and prolongs survival in alphaB-crystallin-based desmin-related cardiomyopathy.
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Ongoing controversies surrounding cardiac remodeling: is it black and white-or rather fifty shades of gray?Molecular basis of physiological heart growth: fundamental concepts and new playersThe ubiquitin-proteasome system and cardiovascular diseaseCrystal Structure of R120G Disease Mutant of Human αB-Crystallin Domain Dimer Shows Closure of a GrooveStaying young at heart: autophagy and adaptation to cardiac agingThe ubiquitin proteasome system in human cardiomyopathies and heart failure.Neprilysin impedes islet amyloid formation by inhibition of fibril formation rather than peptide degradation.Manipulation of death pathways in desmin-related cardiomyopathyTubulin hyperacetylation is adaptive in cardiac proteotoxicity by promoting autophagy.Sustained activation of nuclear erythroid 2-related factor 2/antioxidant response element signaling promotes reductive stress in the human mutant protein aggregation cardiomyopathy in miceAtg7 induces basal autophagy and rescues autophagic deficiency in CryABR120G cardiomyocytes.Desmin-related cardiomyopathy: an unfolding storyProteasome functional insufficiency in cardiac pathogenesis.Genetics of inherited cardiomyopathy.Protein post-translational modifications and misfolding: new concepts in heart failure.Neuromuscular electrical stimulation prevents skeletal muscle dysfunction in adjuvant-induced arthritis rat.Protein quality control and degradation in cardiomyocytes.Divide and conquer: the application of organelle proteomics to heart failure.Do we age because we have mitochondria?Proteotoxicity and cardiac dysfunction.Myofibrillar Myopathies: New Perspectives from Animal Models to Potential Therapeutic Approaches.Cardiomyocyte expression of a polyglutamine preamyloid oligomer causes heart failure.Modeling human protein aggregation cardiomyopathy using murine induced pluripotent stem cells.Proteostasis in cardiac health and disease.An Unbiased High-Throughput Screen to Identify Novel Effectors That Impact on Cardiomyocyte Aggregate Levels.Upregulation of autophagy genes and the unfolded protein response in human heart failure.Doxycycline attenuates protein aggregation in cardiomyocytes and improves survival of a mouse model of cardiac proteinopathy.Enhanced autophagy ameliorates cardiac proteinopathy.Sumo E2 enzyme UBC9 is required for efficient protein quality control in cardiomyocytes.Aerobic exercise training rescues protein quality control disruption on white skeletal muscle induced by chronic kidney disease in rats.The role of αB-crystallin in skeletal and cardiac muscle tissues.Desmin loss and mitochondrial damage precede left ventricular systolic failure in volume overload heart failure.
P2860
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P2860
Exercise reverses preamyloid oligomer and prolongs survival in alphaB-crystallin-based desmin-related cardiomyopathy.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@ast
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@en
type
label
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@ast
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@en
prefLabel
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@ast
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@en
P2093
P2860
P356
P1476
Exercise reverses preamyloid o ...... desmin-related cardiomyopathy.
@en
P2093
Alina Maloyan
Charles G Glabe
James Gulick
Jeffrey Robbins
Rakez Kayed
P2860
P304
P356
10.1073/PNAS.0609202104
P407
P577
2007-03-26T00:00:00Z