Selective response of various brain cell types during neurodegeneration induced by mild impairment of oxidative metabolism.
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Thiamine nutritional status and depressive symptoms are inversely associated among older Chinese adultsResearch advances at the Institute for Nutritional Sciences at Shanghai, ChinaActivation of double-stranded RNA-activated protein kinase by mild impairment of oxidative metabolism in neuronsNutritional Status Driving Infection by Trypanosoma cruzi: Lessons from Experimental Animals.Thiamine deficiency promotes T cell infiltration in experimental autoimmune encephalomyelitis: the involvement of CCL2Abnormal thiamine-dependent processes in Alzheimer's Disease. Lessons from diabetesADAR2-dependent RNA editing of GluR2 is involved in thiamine deficiency-induced alteration of calcium dynamics.Neuronal MCP-1 mediates microglia recruitment and neurodegeneration induced by the mild impairment of oxidative metabolism.Association of SOD2, a mitochondrial antioxidant enzyme, with gray matter volume shrinkage in alcoholicsA mitocentric view of Alzheimer's disease suggests multi-faceted treatmentsEthanol promotes thiamine deficiency-induced neuronal death: involvement of double-stranded RNA-activated protein kinase.Thiamine deficiency induces endoplasmic reticulum stress in neurons.Excessive α-tocopherol exacerbates microglial activation and brain injury caused by acute ischemic stroke.Vitamin B1 (thiamine) and dementiaAutophagy alleviates neurodegeneration caused by mild impairment of oxidative metabolism.Oxidant-induced changes in mitochondria and calcium dynamics in the pathophysiology of Alzheimer's disease.Thiamine deficiency induces oxidative stress and exacerbates the plaque pathology in Alzheimer's mouse model.Role of thiamine in Alzheimer's disease.Translocation of amyloid precursor protein C-terminal fragment(s) to the nucleus precedes neuronal death due to thiamine deficiency-induced mild impairment of oxidative metabolism.Responses of the mitochondrial alpha-ketoglutarate dehydrogenase complex to thiamine deficiency may contribute to regional selective vulnerability.Changes in inflammatory processes associated with selective vulnerability following mild impairment of oxidative metabolismAssociations between in vivo neuroimaging and postmortem brain cytokine markers in a rodent model of Wernicke's encephalopathy.PKR downregulation prevents neurodegeneration and β-amyloid production in a thiamine-deficient model.Thiamine deficiency induces endoplasmic reticulum stress and oxidative stress in human neurons derived from induced pluripotent stem cells.Adjuvant thiamine improved standard treatment in patients with major depressive disorder: results from a randomized, double-blind, and placebo-controlled clinical trial.Chronic Ethanol Consumption and Thiamine Deficiency Modulate β-Amyloid Peptide Level and Oxidative Stress in the Brain.
P2860
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P2860
Selective response of various brain cell types during neurodegeneration induced by mild impairment of oxidative metabolism.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Selective response of various ...... rment of oxidative metabolism.
@ast
Selective response of various ...... rment of oxidative metabolism.
@en
type
label
Selective response of various ...... rment of oxidative metabolism.
@ast
Selective response of various ...... rment of oxidative metabolism.
@en
prefLabel
Selective response of various ...... rment of oxidative metabolism.
@ast
Selective response of various ...... rment of oxidative metabolism.
@en
P1476
Selective response of various ...... rment of oxidative metabolism.
@en
P2093
Gary E Gibson
P304
P356
10.1016/J.NEUINT.2003.09.008
P50
P577
2004-07-01T00:00:00Z