The White-Nose Syndrome Transcriptome: Activation of Anti-fungal Host Responses in Wing Tissue of Hibernating Little Brown Myotis.
about
Vitamin B2 as a virulence factor in Pseudogymnoascus destructans skin infectionWinter behavior of bats and the progression of white-nose syndrome in the southeastern United States.White-nose syndrome survivors do not exhibit frequent arousals associated with Pseudogymnoascus destructans infection.Deconstructing the Bat Skin Microbiome: Influences of the Host and the Environment.White-nose syndrome increases torpid metabolic rate and evaporative water loss in hibernating bats.Integrated transcriptomic and metabolomic analysis reveals adaptive changes of hibernating retinas.Body temperatures of hibernating little brown bats reveal pronounced behavioural activity during deep torpor and suggest a fever response during white-nose syndrome.White-nose syndrome pathology grading in Nearctic and Palearctic bats.Pseudogymnoascus destructans transcriptome changes during white-nose syndrome infections.Energy conserving thermoregulatory patterns and lower disease severity in a bat resistant to the impacts of white-nose syndrome.The other white-nose syndrome transcriptome: Tolerant and susceptible hosts respond differently to the pathogen Pseudogymnoascus destructans.Profiling the immunome of little brown myotis provides a yardstick for measuring the genetic response to white-nose syndrome.Transcriptomic Crosstalk between Fungal Invasive Pathogens and Their Host Cells: Opportunities and Challenges for Next-Generation Sequencing Methods.Extreme sensitivity to ultraviolet light in the fungal pathogen causing white-nose syndrome of bats.Food for contagion: synthesis and future directions for studying host-parasite responses to resource shifts in anthropogenic environments.Gene expression differs in susceptible and resistant amphibians exposed to Batrachochytrium dendrobatidis.Immune responses in hibernating little brown myotis (Myotis lucifugus) with white-nose syndrome.Pseudogymnoascus destructans: Causative Agent of White-Nose Syndrome in Bats Is Inhibited by Safe Volatile Organic Compounds.Alterations in the health of hibernating bats under pathogen pressure.Environmentally persistent pathogens present unique challenges for studies of host–pathogen interactions: Reply to Field (2018).Quantification of pathogen levels is necessary to compare responses to pathogen exposure: Comment on Davy et al. “The other white‐nose syndrome transcriptome”.Going to Bat(s) for Studies of Disease ToleranceWhite-nose syndrome is associated with increased replication of a naturally persisting coronaviruses in batsGalleria mellonella as an insect model for P. destructans, the cause of White-nose Syndrome in bats
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P2860
The White-Nose Syndrome Transcriptome: Activation of Anti-fungal Host Responses in Wing Tissue of Hibernating Little Brown Myotis.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@ast
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@en
type
label
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@ast
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@en
prefLabel
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@ast
The White-Nose Syndrome Transc ...... bernating Little Brown Myotis.
@en
P2093
P2860
P921
P1433
P1476
The White-Nose Syndrome Transc ...... ibernating Little Brown Myotis
@en
P2093
DeeAnn M Reeder
Elizabeth J Rogers
Joseph S Johnson
Melissa J Behr
Sophia M Reeder
P2860
P304
P356
10.1371/JOURNAL.PPAT.1005168
P577
2015-10-01T00:00:00Z