Mitochondrial permeability transition pore component cyclophilin D distinguishes nigrostriatal dopaminergic death paradigms in the MPTP mouse model of Parkinson's disease.
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Cyclophilin D Promotes Brain Mitochondrial F1FO ATP Synthase Dysfunction in Aging Mice.Gene-environment interaction models to unmask susceptibility mechanisms in Parkinson's disease.Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction.A single intramuscular injection of rAAV-mediated mutant erythropoietin protects against MPTP-induced parkinsonism.Behavioral deficit, oxidative stress, and mitochondrial dysfunction precede tau pathology in P301S transgenic miceThe mitochondrial calcium regulator cyclophilin D is an essential component of oestrogen-mediated neuroprotection in amyotrophic lateral sclerosisOxidative stress modulates mitochondrial failure and cyclophilin D function in X-linked adrenoleukodystrophyDistinct Nrf2 Signaling Mechanisms of Fumaric Acid Esters and Their Role in Neuroprotection against 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Induced Experimental Parkinson's-Like Disease.Molecular mechanisms of cell death: central implication of ATP synthase in mitochondrial permeability transition.Peptidyl-prolyl isomerases: a full cast of critical actors in cardiovascular diseases.Destabilization of survival factor MEF2D mRNA by neurotoxin in models of Parkinson's disease.Adverse outcome pathways: Application to enhance mechanistic understanding of neurotoxicity.The path from mitochondrial ROS to aging runs through the mitochondrial permeability transition pore.Targeting Nrf2-mediated gene transcription by extremely potent synthetic triterpenoids attenuate dopaminergic neurotoxicity in the MPTP mouse model of Parkinson's disease.Characterization of cyclophilin D in freshwater pearl mussel (Hyriopsis schlegelii).Physical exercise promotes memory capability by enhancing hippocampal mitochondrial functions and inhibiting apoptosis in obesity-induced insulin resistance by high fat diet.An adverse outcome pathway for parkinsonian motor deficits associated with mitochondrial complex I inhibition.Current perspective of mitochondrial biology in Parkinson's disease.
P2860
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P2860
Mitochondrial permeability transition pore component cyclophilin D distinguishes nigrostriatal dopaminergic death paradigms in the MPTP mouse model of Parkinson's disease.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Mitochondrial permeability tra ...... model of Parkinson's disease.
@ast
Mitochondrial permeability tra ...... model of Parkinson's disease.
@en
type
label
Mitochondrial permeability tra ...... model of Parkinson's disease.
@ast
Mitochondrial permeability tra ...... model of Parkinson's disease.
@en
prefLabel
Mitochondrial permeability tra ...... model of Parkinson's disease.
@ast
Mitochondrial permeability tra ...... model of Parkinson's disease.
@en
P2093
P2860
P356
P1476
Mitochondrial permeability tra ...... model of Parkinson's disease.
@en
P2093
Anatoly Starkov
Beverly J Lorenzo
Bobby Thomas
Daniel J Ho
Elizabeth Wille
Lichuan Yang
M Flint Beal
Natalia N Starkova
Noel Y Calingasan
Rebecca Banerjee
P2860
P304
P356
10.1089/ARS.2010.3849
P577
2011-06-16T00:00:00Z