Circulating cell membrane microparticles transfer heme to endothelial cells and trigger vasoocclusions in sickle cell disease.
about
Massive release of extracellular vesicles from cancer cells after photodynamic treatment or chemotherapyHeme-mediated cell activation: the inflammatory puzzle of sickle cell anemia.Extracellular Vesicles in Angiogenesis.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).On the translocation of bacteria and their lipopolysaccharides between blood and peripheral locations in chronic, inflammatory diseases: the central roles of LPS and LPS-induced cell death.Differences of microparticle patterns between sickle cell anemia and hemoglobin SC patients.Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology.Nonhematopoietic Nrf2 dominantly impedes adult progression of sickle cell anemia in mice.New insights into sickle cell disease: mechanisms and investigational therapies.Differential heme release from various hemoglobin redox states and the upregulation of cellular heme oxygenase-1Hepatic Overexpression of Hemopexin Inhibits Inflammation and Vascular Stasis in Murine Models of Sickle Cell Disease.Plasma heme-induced renal toxicity is related to a capillary rarefaction.Hemoglobin-Based Blood Substitutes and the Treatment of Sickle Cell Disease: More Harm than Help?Influence of red blood cell-derived microparticles upon vasoregulation.Prothrombotic aspects of sickle cell disease.Extracellular microvesicle microRNAs in children with sickle cell anaemia with divergent clinical phenotypes.Role of red blood cells in haemostasis and thrombosis.Microparticles in sickle cell anaemia: promise and pitfalls.Targeting Mac-1-mediated leukocyte-RBC interactions uncouples the benefits for acute vaso-occlusion and chronic organ damage.Red cell DAMPs and inflammation.The role of carbon monoxide and heme oxygenase in the prevention of sickle cell disease vaso-occlusive crises.Erythrocytes as Carriers for Drug Delivery in Blood Transfusion and Beyond.Exacerbation of oxidative stress during sickle vaso-occlusive crisis is associated with decreased anti-band 3 autoantibodies rate and increased red blood cell-derived microparticle level: a prospective study.Extracellular vesicles in coronary artery disease.Intravascular hemolysis and the pathophysiology of sickle cell disease.Heme as a Target for Therapeutic Interventions.Oxidative pathways in the sickle cell and beyond.There is blood in the water: hemolysis, hemoglobin, and heme in acute lung injury.The endothelin B receptor plays a crucial role in the adhesion of neutrophils to the endothelium in sickle cell disease.Absorbance and redox based approaches for measuring free heme and free hemoglobin in biological matrices.Haptoglobin: an emerging candidate for phenotypic modulation of sickle cell anemia?Determination of extinction coefficients of human hemoglobin in various redox states.Effects of four antiplatelet/statin combined strategies on immune and inflammatory responses in patients with acute myocardial infarction undergoing pharmacoinvasive strategy: Design and rationale of the B and T Types of Lymphocytes Evaluation in AcOxidized Mutant Human Hemoglobins S and E Induce Oxidative Stress and Bioenergetic Dysfunction in Human Pulmonary Endothelial Cells.Targeting βCys93 in hemoglobin S with an antisickling agent possessing dual allosteric and antioxidant effects.Decrease of externalized phosphatidylserine density on red blood cell-derived microparticles in SCA patients treated with hydroxycarbamide.Physiologic Impact of Circulating RBC Microparticles upon Blood-Vascular Interactions.HO-1hi Patrolling Monocytes Protect against Vaso-occlusion in Sickle Cell Disease.Elevated free hemoglobin and decreased haptoglobin levels are associated with adverse clinical outcomes, unfavorable physiologic measures, and altered inflammatory markers in pediatric cardiac surgery patients.Characterization of Renal Injury and Inflammation in an Experimental Model of Intravascular Hemolysis.
P2860
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P2860
Circulating cell membrane microparticles transfer heme to endothelial cells and trigger vasoocclusions in sickle cell disease.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Circulating cell membrane micr ...... usions in sickle cell disease.
@ast
Circulating cell membrane micr ...... usions in sickle cell disease.
@en
type
label
Circulating cell membrane micr ...... usions in sickle cell disease.
@ast
Circulating cell membrane micr ...... usions in sickle cell disease.
@en
prefLabel
Circulating cell membrane micr ...... usions in sickle cell disease.
@ast
Circulating cell membrane micr ...... usions in sickle cell disease.
@en
P2093
P2860
P50
P1433
P1476
Circulating cell membrane micr ...... usions in sickle cell disease.
@en
P2093
Alain Tedgui
Christina Barja-Fidalgo
Claire Larroque
Dominique Charue
François Lionnet
Hervé Le Clésiau
Jean-Marie Renard
João A De Moraes
Laurent Kiger
Laurent Loufrani
P2860
P304
P356
10.1182/BLOOD-2014-07-589283
P407
P50
P577
2015-03-31T00:00:00Z