Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology. - Wikidata - awgldk - TriplyDB

Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology.

Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology.

http://www.wikidata.org/entity/Q36596850

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Secondhand Smoke Is an Important Modifiable Risk Factor in Sickle Cell Disease: A Review of the Current Literature and Areas for Future Research Inflammation in Sickle Cell Disease: Differential and Down-Expressed Plasma Levels of Annexin A1 Protein Potential therapeutic action of nitrite in sickle cell disease HbS Binding to GP1bα Activates Platelets in Sickle Cell Disease NNKTT120, an anti-iNKT cell monoclonal antibody, produces rapid and sustained iNKT cell depletion in adults with sickle cell disease Electroacupuncture in conscious free-moving mice reduces pain by ameliorating peripheral and central nociceptive mechanisms.ARQ 092, an orally-available, selective AKT inhibitor, attenuates neutrophil-platelet interactions in sickle cell disease.Prothrombotic aspects of sickle cell disease.Emerging point-of-care technologies for sickle cell disease screening and monitoring.Pediatric sickle cell disease: past successes and future challenges.Targeting Mac-1-mediated leukocyte-RBC interactions uncouples the benefits for acute vaso-occlusion and chronic organ damage.Association of circulating transcriptomic profiles with mortality in sickle cell disease.Downstream Regulatory Element Antagonist Modulator (DREAM), a target for anti-thrombotic agents.Hydroxyurea treatment effect on children with sickle cell disease and obstructive sleep apnea.Intravascular hemolysis and the pathophysiology of sickle cell disease.Gene Therapy for β-Hemoglobinopathies.Prasugrel hydrochloride for the treatment of sickle cell disease.Ser/Thr protein kinase Bβ-NADPH oxidase 2 signaling in thromboinflammation.The endothelin B receptor plays a crucial role in the adhesion of neutrophils to the endothelium in sickle cell disease.Oral tetrahydrouridine and decitabine for non-cytotoxic epigenetic gene regulation in sickle cell disease: A randomized phase 1 study.Depletion of HDL3 high density lipoprotein and altered functionality of HDL2 in blood from sickle cell patients.Placenta growth factor mediated gene regulation in sickle cell disease.Extracellular fluid tonicity impacts sickle red blood cell deformability and adhesion.Airway inflammation in sickle cell disease-A translational perspective.Calpain-1 regulates platelet function in a humanized mouse model of sickle cell disease.Inflammatory mediators in sickle cell anaemia highlight the difference between steady state and crisis in paediatric patients.Sickle cell disease: a malady beyond a hemoglobin defect in cerebrovascular disease.Population pharmacokinetics of cefotaxime and dosage recommendations in children with sickle cell disease.HO-1hi Patrolling Monocytes Protect against Vaso-occlusion in Sickle Cell Disease.In situ generation, metabolism and immunomodulatory signaling actions of nitro-conjugated linoleic acid in a murine model of inflammation.Safety and efficacy of plerixafor dose escalation for the mobilization of CD34+ hematopoietic progenitor cells in patients with sickle cell disease: interim results.Developing new pharmacotherapeutic approaches to treating sickle-cell disease.From total blood exchange to erythrocytapheresis and back to treat complications of sickle cell disease.Nitric oxide pathology and therapeutics in sickle cell disease.Massive Ovarian Edema in a Girl with Hemoglobin SC Disease.A common molecular signature of patients with sickle cell disease revealed by microarray meta-analysis and a genome-wide association study Simultaneous polymerization and adhesion under hypoxia in sickle cell disease The role of neutrophils in thrombosis The platelet NLRP3 inflammasome is upregulated in sickle cell disease via HMGB1/TLR4 and Bruton tyrosine kinase Synergistic Integration of Laboratory and Numerical Approaches in Studies of the Biomechanics of Diseased Red Blood Cells

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Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology.

http://www.wikidata.org/entity/Q36596850

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Chunliang Xu

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Dachuan Zhang

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Deepa Manwani

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Paul S Frenette

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P2860

Hydroxyurea induces fetal hemoglobin by the nitric oxide-dependent activation of soluble guanylyl cyclase

Platelet TLR4 activates neutrophil extracellular traps to ensnare bacteria in septic blood

Phase 1 study of the E-selectin inhibitor GMI 1070 in patients with sickle cell anemia

Adenosine A2A receptors induced on iNKT and NK cells reduce pulmonary inflammation and injury in mice with sickle cell disease.

P-selectin-mediated platelet-neutrophil aggregate formation activates neutrophils in mouse and human sickle cell disease.

NKT cells mediate pulmonary inflammation and dysfunction in murine sickle cell disease through production of IFN-gamma and CXCR3 chemokines.

Platelet-neutrophil-interactions: linking hemostasis and inflammation.

Primary role for adherent leukocytes in sickle cell vascular occlusion: a new paradigm

Heterotypic interactions enabled by polarized neutrophil microdomains mediate thromboinflammatory injury

CXCL1 and its receptor, CXCR2, mediate murine sickle cell vaso-occlusion during hemolytic transfusion reactions

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pathophysiology

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