Human ApoA-II inhibits the hydrolysis of HDL triglyceride and the decrease of HDL size induced by hypertriglyceridemia and cholesteryl ester transfer protein in transgenic mice
about
Human apolipoprotein C-I accounts for the ability of plasma high density lipoproteins to inhibit the cholesteryl ester transfer protein activityFunctional lecithin:cholesterol acyltransferase deficiency and high density lipoprotein deficiency in transgenic mice overexpressing human apolipoprotein A-IIDramatically decreased high density lipoprotein cholesterol, increased remnant clearance, and insulin hypersensitivity in apolipoprotein A-II knockout mice suggest a complex role for apolipoprotein A-II in atherosclerosis susceptibilityMolecular cloning and expression of lipid transfer inhibitor protein reveals its identity with apolipoprotein FThe effect of weight loss on HDL subfractions and LCAT activity in two genotypes of APOA-II -265T>C polymorphismTriglyceride enrichment of HDL enhances in vivo metabolic clearance of HDL apo A-I in healthy men.Role of the tumor suppressor IQGAP2 in metabolic homeostasis: Possible link between diabetes and cancer.Effect of rosiglitazone on HDL metabolism in subjects with metabolic syndrome and low HDL.Evaluating the association of APOA2 polymorphism with insulin resistance in adolescents.Polymorphisms of mouse apolipoprotein A-II alter its physical and functional nature.High yield expression and purification of recombinant human apolipoprotein A-II in Escherichia coliAPOA2 Polymorphism in Relation to Obesity and Lipid Metabolism.Treatment of metabolic syndrome with ankaflavin, a secondary metabolite isolated from the edible fungus Monascus spp.Apolipoprotein AII is a regulator of very low density lipoprotein metabolism and insulin resistance.Apolipoprotein A-II modulates the binding and selective lipid uptake of reconstituted high density lipoprotein by scavenger receptor BI.Effects of cholesteryl ester transfer protein inhibition on apolipoprotein A-II-containing HDL subspecies and apolipoprotein A-II metabolism.Apolipoprotein CI deficiency markedly augments plasma lipoprotein changes mediated by human cholesteryl ester transfer protein (CETP) in CETP transgenic/ApoCI-knocked out mice.Mechanisms of HDL deficiency in mice overexpressing human apoA-II.Overexpression of apoC-I in apoE-null mice: severe hypertriglyceridemia due to inhibition of hepatic lipase.Mechanisms mediating insulin resistance in transgenic mice overexpressing mouse apolipoprotein A-II.Global proteomic profiling reveals altered proteomic signature in schizophrenia serum.Apolipoprotein A-II induces acute-phase response associated AA amyloidosis in mice through conformational changes of plasma lipoprotein structure.
P2860
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P2860
Human ApoA-II inhibits the hydrolysis of HDL triglyceride and the decrease of HDL size induced by hypertriglyceridemia and cholesteryl ester transfer protein in transgenic mice
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@ast
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@en
type
label
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@ast
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@en
prefLabel
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@ast
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@en
P2093
P2860
P356
P1476
Human ApoA-II inhibits the hyd ...... fer protein in transgenic mice
@en
P2093
P2860
P304
P356
10.1172/JCI117614
P407
P577
1994-12-01T00:00:00Z