Immunopathogenic role of T-cell subsets in Borna disease virus-induced progressive encephalitis.
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Borna disease virus nucleoprotein (p40) is a major target for CD8(+)-T-cell-mediated immune responseVirus-specific CD4+ T cells eliminate borna disease virus from the brain via induction of cytotoxic CD8+ T cellsImmunological and PCR analyses for Borna disease virus in psychiatric patients and blood donors in JapanAre anti-ganglioside antibodies associated with proventricular dilatation disease in birds?A functional role for neutralizing antibodies in Borna disease: influence on virus tropism outside the central nervous systemTrafficking of immune cells in the central nervous systemPathogenesis of borna disease virus: granulocyte fractions of psychiatric patients harbor infectious virus in the absence of antiviral antibodies.Neutralizing antibodies in persistent borna disease virus infection: prophylactic effect of gp94-specific monoclonal antibodies in preventing encephalitisCD8(+) T lymphocytes mediate Borna disease virus-induced immunopathology independently of perforinHigh-dose Borna disease virus infection induces a nucleoprotein-specific cytotoxic T-lymphocyte response and prevention of immunopathology.Two major histocompatibility complex class I-restricted epitopes of the Borna disease virus p10 protein identified by cytotoxic T lymphocytes induced by DNA-based immunizationGlutamate and lipid metabolic perturbation in the hippocampi of asymptomatic borna disease virus-infected horses.Induction of degenerative brain lesions after adoptive transfer of brain lymphocytes from Borna disease virus-infected rats: presence of CD8+ T cells and perforin mRNAT cell ignorance in mice to Borna disease virus can be overcome by peripheral expression of the viral nucleoproteinT cells cause acute immunopathology and are required for long-term survival in mouse adenovirus type 1-induced encephalomyelitisProtective and Pathological Immunity during Central Nervous System Infections.Borna disease virus-induced neurological disorder in mice: infection of neonates results in immunopathology.Identification of the immunodominant H-2K(k)-restricted cytotoxic T-cell epitope in the Borna disease virus nucleoprotein.Prevention of virus persistence and protection against immunopathology after Borna disease virus infection of the brain by a novel Orf virus recombinant.Pathogenic potential of borna disease virus lacking the immunodominant CD8 T-cell epitope.
P2860
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P2860
Immunopathogenic role of T-cell subsets in Borna disease virus-induced progressive encephalitis.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@ast
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@en
type
label
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@ast
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@en
prefLabel
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@ast
Immunopathogenic role of T-cel ...... uced progressive encephalitis.
@en
P2093
P2860
P1433
P1476
Immunopathogenic role of T-cel ...... duced progressive encephalitis
@en
P2093
P2860
P304
P407
P577
1995-02-01T00:00:00Z