Autoantibodies developing to myeloperoxidase and proteinase 3 in systemic vasculitis stimulate neutrophil cytotoxicity toward cultured endothelial cells
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Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in miceCurrent and future prospects in the management of granulomatosis with polyangiitis (Wegener's granulomatosis)Lack of effect of antineutrophil cytoplasmic antibodies associated with ulcerative colitis on superoxide anion production from neutrophilsAnimal models in ANCA-vasculitisAntineutrophil cytoplasm antibodies and vasculitisPulmonary vasculitisApoptosis of endothelial cells induced by the neutrophil serine proteases proteinase 3 and elastaseTheodore E. Woodward Award. Do ANCA cause small vessel vasculitis?PR3-ANCA in Wegener's granulomatosis prime human mononuclear cells for enhanced activation via TLRs and NOD1/2Anti-neutrophil cytoplasmic antibodies (ANCA) from patients with systemic vasculitis recognize restricted epitopes of proteinase 3 involving the catalytic siteAmphipathic variable region heavy chain peptides derived from monoclonal human Wegener's anti-PR3 antibodies stimulate lymphocytes from patients with Wegener's granulomatosis and microscopic polyangiitisTh2 dominance in nasal mucosa in patients with Wegener's granulomatosisPatients with systemic vasculitis have increased levels of autoantibodies against oxidized LDLThe immunopathology of ANCA-associated vasculitisSmall vessel vasculitis of the lungSulfhydryl modification induces calcium entry through IP₃-sensitive store-operated pathway in activation-dependent human neutrophils.The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies.Prognostic factors for hospital mortality and ICU admission in patients with ANCA-related pulmonary vasculitis.Monocyte activation in patients with Wegener's granulomatosis.Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodiesComparative investigation of respiratory tract involvement in granulomatosis with polyangiitis between PR3-ANCA positive and MPO-ANCA positive cases: a retrospective cohort study.Benefits of an expanded use of plasma exchange for anti-neutrophil cytoplasmic antibody-associated vasculitis within a dedicated clinical service.B cell epitope specificity in ANCA-associated vasculitis: does it matter?Decreased CXCR1 and CXCR2 expression on neutrophils in anti-neutrophil cytoplasmic autoantibody-associated vasculitides potentially increases neutrophil adhesion and impairs migration.Antineutrophil cytoplasmic antibodies induce monocyte IL-8 release. Role of surface proteinase-3, alpha1-antitrypsin, and Fcgamma receptors.In vivo approaches to investigate ANCA-associated vasculitis: lessons and limitations.Pathogenesis of antineutrophil cytoplasmic autoantibody-associated small-vessel vasculitis.Accessory left gastric artery aneurysms in granulomatosis with polyangiitis: a case report and literature review.Antibodies to proteinase 3 prime human oral, lung, and kidney epithelial cells to secrete proinflammatory cytokines upon stimulation with agonists to various Toll-like receptors, NOD1, and NOD2.Autoantibodies to myeloperoxidase aggravate mild anti-glomerular-basement-membrane-mediated glomerular injury in the rat.Systemic injection of products of activated neutrophils and H2O2 in myeloperoxidase-immunized rats leads to necrotizing vasculitis in the lungs and gut.Myeloperoxidase-specific plasma cell depletion by bortezomib protects from anti-neutrophil cytoplasmic autoantibodies-induced glomerulonephritis.Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.Wegener's granulomatosis: anti-proteinase 3 antibodies are potent inductors of human endothelial cell signaling and leakage response.Selective killing of B-cell hybridomas targeting proteinase 3, Wegener's autoantigen.T lymphocyte responses to anti-neutrophil cytoplasmic autoantibody (ANCA) antigens are present in patients with ANCA-associated systemic vasculitis and persist during disease remission.Epitope specificity of myeloperoxidase antibodies: identification of candidate human immunodominant epitopes.Impaired granulocyte oxidative burst and decreased expression of leucocyte adhesion molecule-1 (LAM-1) in patients with Wegener's granulomatosis.
P2860
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P2860
Autoantibodies developing to myeloperoxidase and proteinase 3 in systemic vasculitis stimulate neutrophil cytotoxicity toward cultured endothelial cells
description
1992 nî lūn-bûn
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1992年の論文
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1992年論文
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1992年論文
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1992年論文
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1992年論文
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1992年論文
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1992年论文
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name
Autoantibodies developing to m ...... ard cultured endothelial cells
@ast
Autoantibodies developing to m ...... ard cultured endothelial cells
@en
type
label
Autoantibodies developing to m ...... ard cultured endothelial cells
@ast
Autoantibodies developing to m ...... ard cultured endothelial cells
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prefLabel
Autoantibodies developing to m ...... ard cultured endothelial cells
@ast
Autoantibodies developing to m ...... ard cultured endothelial cells
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P2093
P2860
P921
P1476
Autoantibodies developing to m ...... ard cultured endothelial cells
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P2093
P2860
P304
P407
P577
1992-08-01T00:00:00Z