Expression of transforming growth factor-beta 1 and its relation to endomysial fibrosis in progressive muscular dystrophy.
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The latent transforming growth factor beta binding protein (LTBP) familyThe quasi-parallel lives of satellite cells and atrophying musclePrevalence and distribution of regional scar in dysfunctional myocardial segments in Duchenne muscular dystrophy.MicroRNA-29 overexpression by adeno-associated virus suppresses fibrosis and restores muscle function in combination with micro-dystrophinA Reduction in Selenoprotein S Amplifies the Inflammatory Profile of Fast-Twitch Skeletal Muscle in the mdx Dystrophic Mouse.Angiotensin II type 1 receptor blockade attenuates TGF-beta-induced failure of muscle regeneration in multiple myopathic states.A Wnt-TGFβ2 axis induces a fibrogenic program in muscle stem cells from dystrophic mice.Follistatin improves skeletal muscle healing after injury and disease through an interaction with muscle regeneration, angiogenesis, and fibrosis.Biological approaches to improve skeletal muscle healing after injury and diseaseExpression of transforming growth factor-beta 1 in dystrophic patient muscles correlates with fibrosis. Pathogenetic role of a fibrogenic cytokine.Transforming Growth Factor Beta (TGF-β) Is a Muscle Biomarker of Disease Progression in ALS and Correlates with Smad Expression.Tyrosine 705 Phosphorylation of STAT3 Is Associated with Phenotype Severity in TGFβ1 Transgenic Mice.Genome-wide Mechanosensitive MicroRNA (MechanomiR) Screen Uncovers Dysregulation of Their Regulatory Networks in the mdm Mouse Model of Muscular Dystrophy.Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy.Modifying muscular dystrophy through transforming growth factor-β.Wasting mechanisms in muscular dystrophy.Klotho gene silencing promotes pathology in the mdx mouse model of Duchenne muscular dystrophy.Tissue-resident mesenchymal stem/progenitor cells in skeletal muscle: collaborators or saboteurs?Angiotensin II type 1 receptor antagonists alleviate muscle pathology in the mouse model for laminin-α2-deficient congenital muscular dystrophy (MDC1A).Myofiber-specific inhibition of TGFβ signaling protects skeletal muscle from injury and dystrophic disease in mice.
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P2860
Expression of transforming growth factor-beta 1 and its relation to endomysial fibrosis in progressive muscular dystrophy.
description
1994 nî lūn-bûn
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1994年の論文
@ja
1994年学术文章
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1994年学术文章
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1994年学术文章
@zh-cn
1994年学术文章
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1994年学术文章
@zh-my
1994年学术文章
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1994年學術文章
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1994年學術文章
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name
Expression of transforming gro ...... rogressive muscular dystrophy.
@ast
Expression of transforming gro ...... rogressive muscular dystrophy.
@en
type
label
Expression of transforming gro ...... rogressive muscular dystrophy.
@ast
Expression of transforming gro ...... rogressive muscular dystrophy.
@en
prefLabel
Expression of transforming gro ...... rogressive muscular dystrophy.
@ast
Expression of transforming gro ...... rogressive muscular dystrophy.
@en
P2093
P2860
P1476
Expression of transforming gro ...... progressive muscular dystrophy
@en
P2093
Kanazawa I
Miyazono K
Yamazaki M
P2860
P304
P407
P577
1994-02-01T00:00:00Z