Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
about
Prevention and treatment for cardiac complications in Duchenne and Becker muscular dystrophy and X-linked dilated cardiomyopathyPrevention and treatment for cardiac complications in Duchenne and Becker muscular dystrophyBrain dystrophin-glycoprotein complex: persistent expression of beta-dystroglycan, impaired oligomerization of Dp71 and up-regulation of utrophins in animal models of muscular dystrophyMetabogenic and Nutriceutical Approaches to Address Energy Dysregulation and Skeletal Muscle Wasting in Duchenne Muscular DystrophyInhibitory control over Ca(2+) sparks via mechanosensitive channels is disrupted in dystrophin deficient muscle but restored by mini-dystrophin expressionMorphogenesis of Plasmodium zoites is uncoupled from tensile strength.IMC1b is a putative membrane skeleton protein involved in cell shape, mechanical strength, motility, and infectivity of malaria ookinetesGenetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophyDysbindin, a novel coiled-coil-containing protein that interacts with the dystrobrevins in muscle and brainPregnancy-induced amelioration of muscular dystrophy phenotype in mdx mice via muscle membrane stabilization effect of glucocorticoid¹³C NMR metabolomics: applications at natural abundanceA malaria membrane skeletal protein is essential for normal morphogenesis, motility, and infectivity of sporozoitesMembrane blebbing as an assessment of functional rescue of dysferlin-deficient human myotubes via nonsense suppression.Palmitoylation of Plasmodium alveolins promotes cytoskeletal function.Long-term treatment with naproxcinod significantly improves skeletal and cardiac disease phenotype in the mdx mouse model of dystrophyMechanical and electrophysiological properties of the sarcolemma of muscle fibers in two murine models of muscle dystrophy: col6a1-/- and mdxCombinatorial blockade of calcineurin and CD28 signaling facilitates primary and secondary therapeutic gene transfer by adenovirus vectors in dystrophic (mdx) mouse muscles.Dystrophin and utrophin: genetic analyses of their role in skeletal muscle.Absence of aquaporin-4 in skeletal muscle alters proteins involved in bioenergetic pathways and calcium handling.Mechanisms of muscle weakness in muscular dystrophyDisease-causing missense mutations in actin binding domain 1 of dystrophin induce thermodynamic instability and protein aggregation.Mechanical perturbation elicits a phenotypic difference between Dictyostelium wild-type cells and cytoskeletal mutants.Ca(2+) influx and opening of Ca(2+)-activated K(+) channels in muscle fibers from control and mdx mice.Ex vivo stretch reveals altered mechanical properties of isolated dystrophin-deficient hearts.Role of nitric oxide in the pathogenesis of muscular dystrophies: a "two hit" hypothesis of the cause of muscle necrosis.Malaria IMC1 membrane skeleton proteins operate autonomously and participate in motility independently of cell shape.Sarcospan reduces dystrophic pathology: stabilization of the utrophin-glycoprotein complex.Internal deletion compromises the stability of dystrophinMuscle degeneration without mechanical injury in sarcoglycan deficiencyExpression of transforming growth factor-beta 1 and its relation to endomysial fibrosis in progressive muscular dystrophy.Regulation of cytosolic calcium in skeletal muscle cells of the mdx mouse under conditions of stress.A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin.Complementary distributions of vinculin and dystrophin define two distinct sarcolemma domains in smooth muscleMechanical function of dystrophin in muscle cellsA new model for the interaction of dystrophin with F-actin.Dystrophin protects the sarcolemma from stresses developed during muscle contractionMembrane-stabilizing copolymers confer marked protection to dystrophic skeletal muscle in vivo.Development of approaches to improve cell survival in myoblast transfer therapy.A novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel.Elusive sources of variability of dystrophin rescue by exon skipping
P2860
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P2860
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
description
1991 nî lūn-bûn
@nan
1991 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@ast
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@en
type
label
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@ast
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@en
prefLabel
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@ast
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@en
P356
P1433
P1476
Decreased osmotic stability of dystrophin-less muscle cells from the mdx mouse.
@en
P2093
P2888
P356
10.1038/349069A0
P407
P577
1991-01-01T00:00:00Z
P6179
1014181583