Ca2+ and mitochondria as substrates for deficits in synaptic plasticity in normal brain ageing.
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The endocannabinoid system in normal and pathological brain ageingWhat do we know about the neurogenic potential of different stem cell types?FK506-binding protein 1b/12.6: a key to aging-related hippocampal Ca2+ dysregulation?Aged rats are hypo-responsive to acute restraint: implications for psychosocial stress in aging.Post hoc pattern matching: assigning significance to statistically defined expression patterns in single channel microarray data.Complex patterns of mitochondrial dynamics in human pancreatic cells revealed by fluorescent confocal imaging.Long-term depression in the hippocampal CA1 area of aged rats, revisited: contribution of temporal constraints related to slice preparationSusceptibility to Calcium Dysregulation during Brain Aging.Aging-related gene expression in hippocampus proper compared with dentate gyrus is selectively associated with metabolic syndrome variables in rhesus monkeys.The importance of being subtle: small changes in calcium homeostasis control cognitive decline in normal aging.mTOR pathway and Ca²⁺ stores mobilization in aged smooth muscle cellsDynamic regulation of mitochondrial function by glucocorticoids.Cell biology of normal brain aging: synaptic plasticity-cell death.Mitochondria and Synaptic Plasticity in the Mature and Aging Nervous System.Calcium homeostasis in aging neurons.cAMP promotes the differentiation of neural progenitor cells in vitro via modulation of voltage-gated calcium channels.Environmental enrichment alters dentate granule cell morphology in oldest-old rat.Ca2+ dysregulation in neurons from transgenic mice expressing mutant presenilin 2.The distribution profile and oxidation states of biometals in APP transgenic mouse brain: dyshomeostasis with age and as a function of the development of Alzheimer's disease.Tau Deletion Prevents Stress-Induced Dendritic Atrophy in Prefrontal Cortex: Role of Synaptic Mitochondria.Hypoxia increases Aβ-induced tau phosphorylation by calpain and promotes behavioral consequences in AD transgenic mice.
P2860
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P2860
Ca2+ and mitochondria as substrates for deficits in synaptic plasticity in normal brain ageing.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@ast
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@en
type
label
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@ast
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@en
prefLabel
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@ast
Ca2+ and mitochondria as subst ...... ticity in normal brain ageing.
@en
P2860
P1476
Ca2+ and mitochondria as subst ...... sticity in normal brain ageing
@en
P2093
A Verkhratsky
E C Toescu
P2860
P304
P356
10.1111/J.1582-4934.2004.TB00273.X
P577
2004-04-01T00:00:00Z