Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
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Sulforaphane as a potential protective phytochemical against neurodegenerative diseasesThe multiple functions of cytochrome c and their regulation in life and death decisions of the mammalian cell: from respiration to apoptosisMitochondrial calcium signalling and cell death: approaches for assessing the role of mitochondrial Ca2+ uptake in apoptosisIon Channels and Oxidative Stress as a Potential Link for the Diagnosis or Treatment of Liver DiseasesMitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategiesBcl-x(L) blocks a mitochondrial inner membrane channel and prevents Ca2+ overload-mediated cell deathOxidative stress in Alzheimer's and Parkinson's diseases: insights from the yeast Saccharomyces cerevisiaeThe science of cerebral ischemia and the quest for neuroprotection: navigating past failure to future successThe suppressor of AAC2 Lethality SAL1 modulates sensitivity of heterologously expressed artemia ADP/ATP carrier to bongkrekate in yeastExamination of physiological function and biochemical disorders in a rat model of prolonged asphyxia-induced cardiac arrest followed by cardio pulmonary bypass resuscitationCyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemiaDifferential NMDA receptor-dependent calcium loading and mitochondrial dysfunction in CA1 vs. CA3 hippocampal neuronsNeonatal Hypoxia Ischaemia: Mechanisms, Models, and Therapeutic Challenges.20-Hydroxyecdysone protects against oxidative stress-induced neuronal injury by scavenging free radicals and modulating NF-κB and JNK pathwaysGABAergic mechanism of propofol toxicity in immature neuronsSrc kinase up-regulates the ERK cascade through inactivation of protein phosphatase 2A following cerebral ischemia.Ethyl pyruvate protects against hypoxic-ischemic brain injury via anti-cell death and anti-inflammatory mechanisms.Safety and tolerability of cyclosporin a in severe traumatic brain injury patients: results from a prospective randomized trialThe Golgi Ca2+-ATPase KlPmr1p function is required for oxidative stress response by controlling the expression of the heat-shock element HSP60 in Kluyveromyces lactis.Calcium and mitochondrial reactive oxygen species generation: how to read the facts.A distinct sequence in the adenine nucleotide translocase from Artemia franciscana embryos is associated with insensitivity to bongkrekate and atypical effects of adenine nucleotides on Ca2+ uptake and sequestration.Sirt3 protects cortical neurons against oxidative stress via regulating mitochondrial Ca2+ and mitochondrial biogenesis.Docosahexaenoic acid: brain accretion and roles in neuroprotection after brain hypoxia and ischemiaEmerging roles of canonical TRP channels in neuronal function.Mitochondrial mechanisms of cell death and neuroprotection in pediatric ischemic and traumatic brain injury.Cyclosporin A preserves mitochondrial function after traumatic brain injury in the immature rat and piglet.Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III.Proteomics in neurodegenerative diseases: Methods for obtaining a closer look at the neuronal proteome.Aging and amyloid beta-induced oxidative DNA damage and mitochondrial dysfunction in Alzheimer's disease: implications for early intervention and therapeutics.Differences in the protein expression levels of Trx2 and Prx3 in the hippocampal CA1 region between adult and aged gerbils following transient global cerebral ischemia.The role of intracellular calcium for the development and treatment of neuroblastoma.Insights into the Molecular Pathogenesis of Activated B-Cell-like Diffuse Large B-Cell Lymphoma and Its Therapeutic Implications.Oxidative stress and aberrant signaling in aging and cognitive decline.Ginkgo biloba extract (EGb 761) prevents the ischemic brain injury-induced decrease in parvalbumin expression.The therapeutic potential of mitochondrial channels in cancer, ischemia-reperfusion injury, and neurodegeneration.The Parkinson's disease gene DJ-1 is also a key regulator of stroke-induced damage.Gingko biloba extract (EGb 761) attenuates ischemic brain injury-induced reduction in Ca(2+) sensor protein hippocalcin.Synergy between docosahexaenoic acid and butyrate elicits p53-independent apoptosis via mitochondrial Ca(2+) accumulation in colonocytes.Docosahexaenoic acid-induced unfolded protein response, cell cycle arrest, and apoptosis in vascular smooth muscle cells are triggered by Ca²⁺-dependent induction of oxidative stressCalcium-dependent mitochondrial function and dysfunction in neurons.
P2860
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P2860
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@ast
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@en
type
label
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@ast
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@en
prefLabel
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@ast
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury.
@en
P1433
P1476
Mitochondrial calcium and oxidative stress as mediators of ischemic brain injury
@en
P2093
Gary Fiskum
P304
P356
10.1016/J.CECA.2004.02.012
P577
2004-09-01T00:00:00Z