Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
about
Inhibition of herpes simplex virus reactivation by dipyridamoleHuman thymidine kinase can functionally replace herpes simplex virus type 1 thymidine kinase for viral replication in mouse sensory ganglia and reactivation from latency upon explant.Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo.Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishment.Translational compensation of a frameshift mutation affecting herpes simplex virus thymidine kinase is sufficient to permit reactivation from latency.Interferons regulate the phenotype of wild-type and mutant herpes simplex viruses in vivo.
P2860
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@ast
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@en
type
label
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@ast
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@en
prefLabel
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@ast
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@en
P2093
P2860
P1433
P1476
Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside
@en
P2093
P2860
P304
P407
P577
1996-02-01T00:00:00Z