Evidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons.
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Herpes simplex virus transactivator VP16 discriminates between HCF-1 and a novel family member, HCF-2.Potential role for luman, the cellular homologue of herpes simplex virus VP16 (alpha gene trans-inducing factor), in herpesvirus latencyExplant-induced reactivation of herpes simplex virus occurs in neurons expressing nuclear cdk2 and cdk4Spontaneous reactivation of herpes simplex virus type 1 in latently infected murine sensory gangliaInhibition of herpes simplex virus reactivation by dipyridamoleNuclear localization of the C1 factor (host cell factor) in sensory neurons correlates with reactivation of herpes simplex virus from latencyTransient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neuronsDe novo synthesis of VP16 coordinates the exit from HSV latency in vivo.A dominant-negative herpesvirus protein inhibits intranuclear targeting of viral proteins: effects on DNA replication and late gene expression.Genetic studies exposing the splicing events involved in herpes simplex virus type 1 latency-associated transcript production during lytic and latent infection.Transcription of the herpes simplex virus 1 genome during productive and quiescent infection of neuronal and nonneuronal cells.The polyserine tract of herpes simplex virus ICP4 is required for normal viral gene expression and growth in murine trigeminal gangliaThe latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivationThe PK domain of the large subunit of herpes simplex virus type 2 ribonucleotide reductase (ICP10) is required for immediate-early gene expression and virus growthRegulation of herpes simplex virus type 1 thymidine kinase gene expression by thyroid hormone receptor in cultured neuronal cells.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Site-directed mutagenesis of large DNA palindromes: construction and in vitro characterization of herpes simplex virus type 1 mutants containing point mutations that eliminate the oriL or oriS initiation functionAn intertypic herpes simplex virus helicase-primase complex associated with a defect in neurovirulence has reduced primase activity.Herpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency.Point mutations in herpes simplex virus type 1 oriL, but not in oriS, reduce pathogenesis during acute infection of mice and impair reactivation from latency.Histone deacetylase inhibitors induce reactivation of herpes simplex virus type 1 in a latency-associated transcript-independent manner in neuronal cellsHerpes simplex virus type 1 origins of DNA replication play no role in the regulation of flanking promoters.Tegument protein control of latent herpesvirus establishment and animation.Effects of thyroid hormone on HSV-1 gene regulation: implications in the control of viral latency and reactivation.Prevalence and abundance of latently transcribed varicella-zoster virus genes in human ganglia.Cellular transcription factors induced in trigeminal ganglia during dexamethasone-induced reactivation from latency stimulate bovine herpesvirus 1 productive infection and certain viral promoters.Quantification of transcripts from the ICP4 and thymidine kinase genes in mouse ganglia latently infected with herpes simplex virusGamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1.Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleosideHerpes simplex virus gene expression in neurons: viral DNA synthesis is a critical regulatory event in the branch point between the lytic and latent pathways.Gene expression during reactivation of herpes simplex virus type 1 from latency in the peripheral nervous system is different from that during lytic infection of tissue culturesComprehensive quantification of herpes simplex virus latency at the single-cell levelThe herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latencyA LAT-associated function reduces productive-cycle gene expression during acute infection of murine sensory neurons with herpes simplex virus type 1.Analysis of bovine herpesvirus 1 transcripts during a primary infection of trigeminal ganglia of cattle.Upregulation of class I major histocompatibility complex gene expression in primary sensory neurons, satellite cells, and Schwann cells of mice in response to acute but not latent herpes simplex virus infection in vivo.Neuron-specific restriction of a herpes simplex virus recombinant maps to the UL5 gene.Effect of the transcription start region of the herpes simplex virus type 1 latency-associated transcript promoter on expression of productively infected neurons in vivo.Secondary herpes simplex virus latent infection in transplanted ganglia.Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia.
P2860
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P2860
Evidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@ast
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@en
type
label
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@ast
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@en
prefLabel
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@ast
Evidence for a novel regulator ...... n trigeminal ganglion neurons.
@en
P2093
P2860
P1433
P1476
Evidence for a novel regulator ...... in trigeminal ganglion neurons
@en
P2093
J Jacobson
M Kosz-Vnenchak
P2860
P304
P407
P577
1993-09-01T00:00:00Z