Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma. - Wikidata - awgldk - TriplyDB

Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

http://www.wikidata.org/entity/Q35862636

about

FGFR3-TACC3 fusion in solid tumors: mini review Clinical association analysis of ependymomas and pilocytic astrocytomas reveals elevated FGFR3 and FGFR1 expression in aggressive ependymomas.TCF12 is mutated in anaplastic oligodendroglioma CNS Anticancer Drug Discovery and Development Conference White Paper.Genetic Landscape of Human Papillomavirus-Associated Head and Neck Cancer and Comparison to Tobacco-Related Tumors.Development of RNA-FISH Assay for Detection of Oncogenic FGFR3-TACC3 Fusion Genes in FFPE Samples Fibroblast growth factor receptor 3 protein is overexpressed in oral and oropharyngeal squamous cell carcinoma.Targeted next-generation sequencing panel (GlioSeq) provides comprehensive genetic profiling of central nervous system tumors.Polycomb dysregulation in gliomagenesis targets a Zfp423-dependent differentiation network.FGFR gene alterations in lung squamous cell carcinoma are potential targets for the multikinase inhibitor nintedanib.FGFR3-TACC3 fusion proteins act as naturally occurring drivers of tumor resistance by functionally substituting for EGFR/ERK signaling.Polymorphous low-grade neuroepithelial tumor of the young (PLNTY): an epileptogenic neoplasm with oligodendroglioma-like components, aberrant CD34 expression, and genetic alterations involving the MAP kinase pathway.FGFR3, as a receptor tyrosine kinase, is associated with differentiated biological functions and improved survival of glioma patients.Target and Agent Prioritization for the Children's Oncology Group-National Cancer Institute Pediatric MATCH Trial.Emerging circulating biomarkers in glioblastoma: promises and challenges.Glioblastoma targeted therapy: updated approaches from recent biological insights.Targeting Signaling Transduction Pathways in Bladder Cancer.Evolving Molecular Genetics of Glioblastoma.The Effect of Molecular Diagnostics on the Treatment of Glioma.Advances in the molecular genetics of gliomas - implications for classification and therapy.Genetic biomarkers of drug response for small-molecule therapeutics targeting the RTK/Ras/PI3K, p53 or Rb pathway in glioblastoma.Further understanding of the pathology of glioma: implications for the clinic.FGFR-TACC gene fusions in human glioma.Biomarkers for precision medicine in bladder cancer.Insights into molecular therapy of glioma: current challenges and next generation blueprint Integrating RNA sequencing into neuro-oncology practice.Clonal evolution of glioblastoma under therapy.Strong FGFR3 staining is a marker for FGFR3 fusions in diffuse gliomas.Unique signalling connectivity of FGFR3-TACC3 oncoprotein revealed by quantitative phosphoproteomics and differential network analysis.A metabolic function of FGFR3-TACC3 gene fusions in cancer.Molecular mechanisms underlying gliomas and glioblastoma pathogenesis revealed by bioinformatics analysis of microarray data.Drug repurposing for the treatment of glioblastoma multiforme.Activating FGFR1 Mutations in Sporadic Pheochromocytomas.Neurological update: gliomas and other primary brain tumours in adults.Diffuse gliomas with FGFR3-TACC3 fusion have characteristic histopathological and molecular features.Current therapeutic approaches to diffuse grade II and III gliomas.Glioblastoma: new therapeutic strategies to address cellular and genomic complexity.Tumor Evolution and Drug Response in Patient-Derived Organoid Models of Bladder Cancer.Comprehensive genomic profiling aids in treatment of a metastatic endometrial cancer.Comprehensive genomic profiling of malignant phyllodes tumors of the breast.

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P2860

Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

http://www.wikidata.org/entity/Q35862636

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

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Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

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type

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Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

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Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

@en

prefLabel

Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

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Detection, Characterization, and Inhibition of FGFR-TACC Fusions in IDH Wild-type Glioma.

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Clinical Cancer Research

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Anna Luisa Di Stefano

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Hayat Belaid

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Jean-Charles Soria

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P2860

Identification of targetable FGFR gene fusions in diverse cancers.

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

The somatic genomic landscape of glioblastoma

Comprehensive molecular characterization of urothelial bladder carcinoma

Circular binary segmentation for the analysis of array-based DNA copy number data

Transforming fusions of FGFR and TACC genes in human glioblastoma.

An integrative approach to reveal driver gene fusions from paired-end sequencing data in cancer.

Tumor heterogeneity is an active process maintained by a mutant EGFR-induced cytokine circuit in glioblastoma.

Comparative assessment of 5 methods (methylation-specific polymerase chain reaction, MethyLight, pyrosequencing, methylation-sensitive high-resolution melting, and immunohistochemistry) to analyze O6-methylguanine-DNA-methyltranferase in a series of

The landscape of kinase fusions in cancer

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