BMI-1, a promising therapeutic target for human cancer. - Wikidata - awgldk - TriplyDB

BMI-1, a promising therapeutic target for human cancer.

BMI-1, a promising therapeutic target for human cancer.

http://www.wikidata.org/entity/Q35870390

about

Polycomb complex protein BMI-1 promotes invasion and metastasis of pancreatic cancer stem cells by activating PI3K/AKT signaling, an ex vivo, in vitro, and in vivo study.BMI1 regulates PRC1 architecture and activity through homo- and hetero-oligomerization.miR-15a/miR-16 down-regulates BMI1, impacting Ub-H2A mediated DNA repair and breast cancer cell sensitivity to doxorubicin Relevance of necroptosis in cancer.PIWI-like protein, HIWI2 is aberrantly expressed in retinoblastoma cells and affects cell-cycle potentially through OTX2.Bmi‑1‑targeting suppresses osteosarcoma aggressiveness through the NF‑κB signaling pathway.Circulating microRNAs as potential biomarkers to detect transformation of Barrett's oesophagus to oesophageal adenocarcinoma.Regulating BMI1 expression via miRNAs promote Mesenchymal to Epithelial Transition (MET) and sensitizes breast cancer cell to chemotherapeutic drug.Targeting of BMI-1 expression by the novel small molecule PTC596 in mantle cell lymphoma.

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P2860

BMI-1, a promising therapeutic target for human cancer.

http://www.wikidata.org/entity/Q35870390

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

BMI-1, a promising therapeutic target for human cancer.

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BMI-1, a promising therapeutic target for human cancer.

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type

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BMI-1, a promising therapeutic target for human cancer.

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BMI-1, a promising therapeutic target for human cancer.

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BMI-1, a promising therapeutic target for human cancer.

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BMI-1, a promising therapeutic target for human cancer.

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P1433

Oncology Letters

P1476

BMI-1, a promising therapeutic target for human cancer.

@en

P2093

Chun-Li Li

http://www.w3.org/2001/XMLSchema#string

Jie Cui

http://www.w3.org/2001/XMLSchema#string

Ke-Jun Nan

http://www.w3.org/2001/XMLSchema#string

L I Jing

http://www.w3.org/2001/XMLSchema#string

Min Jiao

http://www.w3.org/2001/XMLSchema#string

Min-Cong Wang

http://www.w3.org/2001/XMLSchema#string

Tao Wu

http://www.w3.org/2001/XMLSchema#string

P2860

MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1

Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.

miR-9, a MYC/MYCN-activated microRNA, regulates E-cadherin and cancer metastasis.

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

Bmi-1 is a target gene for SALL4 in hematopoietic and leukemic cells

Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells

Tumor suppression by Ink4a-Arf: progress and puzzles

MYC stimulates EZH2 expression by repression of its negative regulator miR-26a

Bmi-1 promotes glioma angiogenesis by activating NF-κB signaling

Bmi-1 determines the proliferative capacity of normal and leukaemic stem cells

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583-588

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10.3892/OL.2015.3361

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2

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