Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
about
Mechanistic insight into the functional and toxic effects of Strophanthidin in the failing human myocardiumCalsequestrin mutations and catecholaminergic polymorphic ventricular tachycardiaSympathetic nervous system overactivity and its role in the development of cardiovascular diseaseGain of function of cardiac ryanodine receptor in a rat model of type 1 diabetesUpregulation of the CaV 1.1-ryanodine receptor complex in a rat model of critical illness myopathyAltered intracellular Ca2+ handling in heart failure.Cardiac adrenergic control and atrial fibrillation.FKBP12 activates the cardiac ryanodine receptor Ca2+-release channel and is antagonised by FKBP12.6.The ryanodine receptor in cardiac physiology and disease.Acute atrial arrhythmogenicity and altered Ca(2+) homeostasis in murine RyR2-P2328S hearts.Catheter-based renal sympathetic denervation significantly inhibits atrial fibrillation induced by electrical stimulation of the left stellate ganglion and rapid atrial pacing.Cardiac and skeletal muscle disorders caused by mutations in the intracellular Ca2+ release channels.Simulation of arrhythmogenic effect of rogue RyRs in failing heart by using a coupled modelInnovative approaches to anti-arrhythmic drug therapy.Mechanisms of sympathetic activation in heart failure.Accelerated sinus rhythm prevents catecholaminergic polymorphic ventricular tachycardia in mice and in patients.Ryanodine receptor allosteric coupling and the dynamics of calcium sparksArrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation.Molecular and electrophysiological bases of catecholaminergic polymorphic ventricular tachycardia.Abnormal Ca(2+) homeostasis, atrial arrhythmogenesis, and sinus node dysfunction in murine hearts modeling RyR2 modificationIn vitro modeling of ryanodine receptor 2 dysfunction using human induced pluripotent stem cells.Illicit organogenesis: Methods and substances of doping and manipulation.Ca2+ signaling in human induced pluripotent stem cell-derived cardiomyocytes (iPS-CM) from normal and catecholaminergic polymorphic ventricular tachycardia (CPVT)-afflicted subjects.CPU228, a derivative of dofetilide, relieves cardiac dysfunction by normalizing FKBP12.6, NADPH oxidase and protein kinase C epsilon in the myocardium.
P2860
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P2860
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@ast
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@en
type
label
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@ast
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@en
prefLabel
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@ast
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@en
P1476
Calstabin deficiency, ryanodine receptors, and sudden cardiac death.
@en
P2093
Andrew R Marks
Stephan E Lehnart
P304
P356
10.1016/J.BBRC.2004.08.032
P407
P577
2004-10-01T00:00:00Z