TNF-induced structural joint damage is mediated by IL-1. - Wikidata - awgldk - TriplyDB

TNF-induced structural joint damage is mediated by IL-1.

TNF-induced structural joint damage is mediated by IL-1.

http://www.wikidata.org/entity/Q35880188

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Animal Models of Bone Loss in Inflammatory Arthritis: from Cytokines in the Bench to Novel Treatments for Bone Loss in the Bedside-a Comprehensive Review Response to peripheral immune stimulation within the brain: magnetic resonance imaging perspective of treatment success A dynamic real time in vivo and static ex vivo analysis of granulomonocytic cell migration in the collagen-induced arthritis model Stable activation of fibroblasts in rheumatic arthritis-causes and consequences Lessons from Microglia Aging for the Link between Inflammatory Bone Disorders and Alzheimer's Disease The synovio-entheseal complex in enthesoarthritis.MicroRNA-140 is expressed in differentiated human articular chondrocytes and modulates interleukin-1 responses The inorganic pyrophosphate transporter ANK preserves the differentiated phenotype of articular chondrocyte.Functional consequences of DECTIN-1 early stop codon polymorphism Y238X in rheumatoid arthritis Bone erosion in rheumatoid arthritis: mechanisms, diagnosis and treatment.A crucial role for tumor necrosis factor receptor 1 in synovial lining cells and the reticuloendothelial system in mediating experimental arthritis Matrix metalloproteinase 8 deficiency in mice exacerbates inflammatory arthritis through delayed neutrophil apoptosis and reduced caspase 11 expression.Constitutively activated NLRP3 inflammasome causes inflammation and abnormal skeletal development in mice.Matrix metalloproteinase-8 deficiency increases joint inflammation and bone erosion in the K/BxN serum-transfer arthritis model.Probiotics protect mice from ovariectomy-induced cortical bone loss.The impact of inflammation on bone mass in children.Bone density and hyperlipidemia: the T-lymphocyte connection.Intra-articular delivery of anti-Hif-2α siRNA by chondrocyte-homing nanoparticles to prevent cartilage degeneration in arthritic mice.Bone Loss Triggered by the Cytokine Network in Inflammatory Autoimmune Diseases.Positive regulators of osteoclastogenesis and bone resorption in rheumatoid arthritis Modulation of interleukin-1β-induced inflammatory responses by a synthetic cationic innate defence regulator peptide, IDR-1002, in synovial fibroblasts An update on the pathology and clinical management of gouty arthritis.E74-like factor 3 (ELF3) impacts on matrix metalloproteinase 13 (MMP13) transcriptional control in articular chondrocytes under proinflammatory stress.Serum interleukin-17 & nitric oxide levels in patients with primary Sjögren's syndrome.Role of proopiomelanocortin-derived peptides and their receptors in the osteoarticular system: from basic to translational research.The gut microbiota regulates bone mass in mice Cytokines profiling by multiplex analysis in experimental arthritis: which pathophysiological relevance for articular versus systemic mediators?IL-1 family cytokines trigger sterile inflammatory disease.ARTD1 regulates osteoclastogenesis and bone homeostasis by dampening NF-κB-dependent transcription of IL-1β MicroRNA-125b regulates the expression of aggrecanase-1 (ADAMTS-4) in human osteoarthritic chondrocytes.Joint remodelling in inflammatory disease.Cell signalling in macrophages, the principal innate immune effector cells of rheumatoid arthritis.Cells of the synovium in rheumatoid arthritis. Chondrocytes.Does gamma-aminobutyric acid (GABA) influence the development of chronic inflammation in rheumatoid arthritis?Cartilage homeostasis in health and rheumatic diseases.Evidence that cytokines play a role in rheumatoid arthritis.Lessons from animal models of arthritis over the past decade.Pathogenesis of joint destruction in rheumatoid arthritis.Effects of inflammatory and anti-inflammatory cytokines on the bone.Temporal cytokine expression and the target organ attributes unravel novel aspects of autoimmune arthritis.

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P2860

TNF-induced structural joint damage is mediated by IL-1.

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TNF-induced structural joint damage is mediated by IL-1.

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TNF-induced structural joint damage is mediated by IL-1.

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TNF-induced structural joint damage is mediated by IL-1.

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TNF-induced structural joint damage is mediated by IL-1.

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Proceedings of the National Academy of Sciences of the United States of America

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TNF-induced structural joint damage is mediated by IL-1.

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Andreas Hess

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Clemens Scheinecker

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Jochen Zwerina

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Joerg Distler

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Juerg Gasser

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Karin Polzer

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Kurt Redlich

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Leo Joosten

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Noreen Pundt

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Oskar Hoffmann

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P2860

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Cytokines in the pathogenesis of rheumatoid arthritis

Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

IL-1 and TNF induction of matrix metalloproteinase-3 by c-Jun N-terminal kinase in trabecular meshwork.

Observational study on efficacy, safety, and drug survival of anakinra in rheumatoid arthritis patients in clinical practice.

M-CSF mediates TNF-induced inflammatory osteolysis

Lessons from animal models of arthritis.

Pathogenesis of bone and cartilage destruction in rheumatoid arthritis.

Open label trial of anakinra in active ankylosing spondylitis over 24 weeks.

The IL-1 family and inflammatory diseases.

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104

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Gerhard Krönke

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