Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2. - Wikidata - awgldk - TriplyDB

Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2.

Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2.

http://www.wikidata.org/entity/Q35889648

about

Sustained activation of SMAD3/SMAD4 by FOXM1 promotes TGF-β-dependent cancer metastasis Epithelial-Mesenchymal Transition (EMT) and Regulation of EMT Factors by Steroid Nuclear Receptors in Breast Cancer: A Review and in Silico Investigation Regulation of Bim in Health and Disease The network of pluripotency, epithelial-mesenchymal transition, and prognosis of breast cancer RUNX2 and the PI3K/AKT axis reciprocal activation as a driving force for tumor progression The Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and Treatment Persistent activation of pancreatic stellate cells creates a microenvironment favorable for the malignant behavior of pancreatic ductal adenocarcinoma Wnt signaling in triple negative breast cancer is associated with metastasis RUNX2 correlates with subtype-specific breast cancer in a human tissue microarray, and ectopic expression of Runx2 perturbs differentiation in the mouse mammary gland.Differential effects of RUNX2 on the androgen receptor in prostate cancer: synergistic stimulation of a gene set exemplified by SNAI2 and subsequent invasiveness.Differences in the transcriptional response to fulvestrant and estrogen deprivation in ER-positive breast cancer Runx2 is a novel regulator of mammary epithelial cell fate in development and breast cancer Meta-analysis of gene expression signatures defining the epithelial to mesenchymal transition during cancer progression Cadherin 6 is a new RUNX2 target in TGF-β signalling pathway The cancer gene WWOX behaves as an inhibitor of SMAD3 transcriptional activity via direct binding RUNX1, a transcription factor mutated in breast cancer, controls the fate of ER-positive mammary luminal cells.MiR-203 downregulation is responsible for chemoresistance in human glioblastoma by promoting epithelial-mesenchymal transition via SNAI2 Genome-wide Runx2 occupancy in prostate cancer cells suggests a role in regulating secretion.Runx2 contributes to the regenerative potential of the mammary epithelium.Overexpression of runt-related transcription factor-2 is associated with advanced tumor progression and poor prognosis in epithelial ovarian cancer RUNX2 and TAZ-dependent signaling pathways regulate soluble E-Cadherin levels and tumorsphere formation in breast cancer cells.miR-218 directs a Wnt signaling circuit to promote differentiation of osteoblasts and osteomimicry of metastatic cancer cells Detection of RUNX2 gene expression in cumulus cells in women undergoing controlled ovarian stimulation.RUNX1 prevents oestrogen-mediated AXIN1 suppression and β-catenin activation in ER-positive breast cancer.The RUNX2 Transcription Factor Negatively Regulates SIRT6 Expression to Alter Glucose Metabolism in Breast Cancer Cells Heparanase promotes myeloma progression by inducing mesenchymal features and motility of myeloma cells Long noncoding RNA GAPLINC promotes invasion in colorectal cancer by targeting SNAI2 through binding with PSF and NONO.Runx2 activates PI3K/Akt signaling via mTORC2 regulation in invasive breast cancer cells.Antagonizing miR-218-5p attenuates Wnt signaling and reduces metastatic bone disease of triple negative breast cancer cells.The Expression and Functional Significance of Runx2 in Hepatocellular Carcinoma: Its Role in Vasculogenic Mimicry and Epithelial-Mesenchymal Transition.The RUNX family in breast cancer: relationships with estrogen signaling.RUNX2 in mammary gland development and breast cancer.RUNX family: Regulation and diversification of roles through interacting proteins.The RUNX family: developmental regulators in cancer.Analyzing the differentially expressed genes and pathway cross-talk in aggressive breast cancer.Hormone-sensing mammary epithelial progenitors: emerging identity and hormonal regulation.Runx2-I is an Early Regulator of Epithelial-Mesenchymal Cell Transition in the Chick Embryo.Relationship between RUNX1 and AXIN1 in ER-negative versus ER-positive Breast Cancer.The Emerging Roles of RUNX Transcription Factors in Epithelial-Mesenchymal Transition.MicroRNA-23b is an independent prognostic marker and suppresses ovarian cancer progression by targeting runt-related transcription factor-2.

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Regulation of breast cancer metastasis by Runx2 and estrogen signaling: the role of SNAI2.

http://www.wikidata.org/entity/Q35889648

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2011 nî lūn-bûn

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Regulation of breast cancer me ...... signaling: the role of SNAI2.

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Breast Cancer Research

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Regulation of breast cancer me ...... signaling: the role of SNAI2.

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Baruch Frenkel

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Debu Tripathy

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Gillian H Little

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Michael Kahn

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Nyam-Osor Chimge

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Sanjeev K Baniwal

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Yi-bu Chen

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P2860

SLUG: a new target of lymphoid enhancer factor-1 in human osteoblasts

Genes that mediate breast cancer metastasis to the brain

Latent bone metastasis in breast cancer tied to Src-dependent survival signals

Modulation of Runx2 activity by estrogen receptor-alpha: implications for osteoporosis and breast cancer

Epithelial-mesenchymal transitions in development and disease

Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

The snail superfamily of zinc-finger transcription factors

Runx2 controls a feed-forward loop between androgen and prolactin-induced protein (PIP) in stimulating T47D cell proliferation

Regulation of adult bone turnover by sex steroids

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