53BP1 deficiency combined with telomere dysfunction activates ATR-dependent DNA damage response.
about
Human single-stranded DNA binding proteins are essential for maintaining genomic stabilityReplication stress in Mammalian cells and its consequences for mitosisTopoIIα prevents telomere fragility and formation of ultra thin DNA bridges during mitosis through TRF1-dependent binding to telomeresHuman RECQL1 participates in telomere maintenanceCrosstalk between telomere maintenance and radiation effects: A key player in the process of radiation-induced carcinogenesis.Essential role for the TRF2 telomere protein in adult skin homeostasisExpression of the genetic suppressor element 24.2 (GSE24.2) decreases DNA damage and oxidative stress in X-linked dyskeratosis congenita cells.ATM Inhibition Potentiates Death of Androgen Receptor-inactivated Prostate Cancer Cells with Telomere Dysfunction.RIF1 counteracts BRCA1-mediated end resection during DNA repairAging Effects on Cardiac Progenitor Cell Physiology.DNA Replication Origins and Fork Progression at Mammalian Telomeres
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P2860
53BP1 deficiency combined with telomere dysfunction activates ATR-dependent DNA damage response.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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2012年论文
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name
53BP1 deficiency combined with ...... dependent DNA damage response.
@ast
53BP1 deficiency combined with ...... dependent DNA damage response.
@en
type
label
53BP1 deficiency combined with ...... dependent DNA damage response.
@ast
53BP1 deficiency combined with ...... dependent DNA damage response.
@en
prefLabel
53BP1 deficiency combined with ...... dependent DNA damage response.
@ast
53BP1 deficiency combined with ...... dependent DNA damage response.
@en
P2860
P356
P1476
53BP1 deficiency combined with ...... dependent DNA damage response.
@en
P2093
Juana M Flores
Paula Martínez
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P304
P356
10.1083/JCB.201110124
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P577
2012-04-01T00:00:00Z