A crucial role for thiol antioxidants in estrogen-deficiency bone loss. - Wikidata - awgldk - TriplyDB

A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

http://www.wikidata.org/entity/Q35919837

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PAMM: a redox regulatory protein that modulates osteoclast differentiation Pathogenesis of osteoporosis: concepts, conflicts, and prospects N-acetylcysteine -- passe-partout or much ado about nothing?Regulation of human osteoclast differentiation by thioredoxin binding protein-2 and redox-sensitive signaling From estrogen-centric to aging and oxidative stress: a revised perspective of the pathogenesis of osteoporosis The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone Bone loss caused by iron overload in a murine model: importance of oxidative stress The role of vitamin E in reversing bone loss.FoxO proteins restrain osteoclastogenesis and bone resorption by attenuating H2O2 accumulation.FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in mice FoxO1 is a positive regulator of bone formation by favoring protein synthesis and resistance to oxidative stress in osteoblasts Osteocyte apoptosis and control of bone resorption following ovariectomy in mice Antioxidant Effect of Fructus Ligustri Lucidi Aqueous Extract in Ovariectomized Rats Is Mediated through Nox4-ROS-NF-κB Pathway Areca nut extract protects against ovariectomy-induced osteoporosis in mice.Sitagliptin, An Anti-diabetic Drug, Suppresses Estrogen Deficiency-Induced OsteoporosisIn Vivo and Inhibits RANKL-Induced Osteoclast Formation and Bone Resorption In Vitro.Oxidative stress and gamma radiation-induced cancellous bone loss with musculoskeletal disuse.Ascorbate synthesis pathway: dual role of ascorbate in bone homeostasis.Mitochondrial mechanisms of estrogen neuroprotection.Daidzein prevents the increase in CD4+CD28null T cells and B lymphopoesis in ovariectomized mice: a key mechanism for anti-osteoclastogenic effect.Effects of Nrf2 deficiency on bone microarchitecture in an experimental model of osteoporosis TRP14 inhibits osteoclast differentiation via its catalytic activity T cells: critical bone regulators in health and disease.The immune system and bone.Oxidative stress stimulates apoptosis and activates NF-kappaB in osteoblastic cells via a PKCbeta/p66shc signaling cascade: counter regulation by estrogens or androgens.Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.Evaluation of oxidative stress parameters and urinary deoxypyridinoline levels in geriatric patients with osteoporosis Vitamin C prevents hypogonadal bone loss ATP6v0d2 deficiency increases bone mass, but does not influence ovariectomy-induced bone loss.Estrogens and Androgens in Skeletal Physiology and Pathophysiology.Estrogen deficiency and bone loss: an inflammatory tale.Olive oil in the prevention and treatment of osteoporosis after artificial menopause Inhibition of NADPH oxidases prevents chronic ethanol-induced bone loss in female rats.The association between oxidative stress and bone mineral density according to menopausal status of Korean women Piper sarmentosum enhances fracture healing in ovariectomized osteoporotic rats: a radiological study.Skeletal involution by age-associated oxidative stress and its acceleration by loss of sex steroids Unraveling the role of FoxOs in bone--insights from mouse models.Caspase-2 maintains bone homeostasis by inducing apoptosis of oxidatively-damaged osteoclasts.A role for ethanol-induced oxidative stress in controlling lineage commitment of mesenchymal stromal cells through inhibition of Wnt/beta-catenin signaling New insights into the functional role of the rheumatoid arthritis shared epitope.Effects of ovariectomy and ascorbic acid supplement on oxidative stress parameters and bone mineral density in rats

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P2860

A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

http://www.wikidata.org/entity/Q35919837

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2003 nî lūn-bûn

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2003年論文

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2003年论文

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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Journal of Clinical Investigation

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A crucial role for thiol antioxidants in estrogen-deficiency bone loss.

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Barrie Kirstein

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Christopher J Jagger

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Geoffrey A Partington

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Jenny M Lean

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Julie T Davies

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Karen Fuller

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Timothy J Chambers

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Zoë L Urry

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P2860

Free radicals in the physiological control of cell function

Oxidants, oxidative stress and the biology of ageing

Premature aging in mice deficient in DNA repair and transcription

Reactive oxygen species, antioxidants, and the mammalian thioredoxin system

p53 mutant mice that display early ageing-associated phenotypes

Estrogen decreases osteoclast formation by down-regulating receptor activator of NF-kappa B ligand (RANKL)-induced JNK activation.

Estrogen prevents oxidative stress-induced endothelial cell apoptosis in rats.

Defective bone formation and anabolic response to exogenous estrogen in mice with targeted disruption of endothelial nitric oxide synthase.

Up-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.

Glutathione-ascorbic acid antioxidant system in animals.

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10.1172/JCI200318859

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193670

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