The yellow fluorescent protein (YFP-H) mouse reveals neuroprotection as a novel mechanism underlying chondroitinase ABC-mediated repair after spinal cord injury.
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Immunohistochemical Distribution of PlexinA4 in the Adult Rat Central Nervous System.Reorganization of Intact Descending Motor Circuits to Replace Lost Connections After InjuryCorticospinal reorganization after spinal cord injuryCortical overexpression of neuronal calcium sensor-1 induces functional plasticity in spinal cord following unilateral pyramidal tract injury in ratThe lateral thoracic nerve and the cutaneous maximus muscle--a novel in vivo model system for nerve degeneration and regeneration studies.A neural extracellular matrix-based method for in vitro hippocampal neuron culture and dopaminergic differentiation of neural stem cells.Crmp4 deletion promotes recovery from spinal cord injury by neuroprotection and limited scar formation.Integrity of cortical perineuronal nets influences corticospinal tract plasticity after spinal cord injury.Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.BDNF-hypersecreting human mesenchymal stem cells promote functional recovery, axonal sprouting, and protection of corticospinal neurons after spinal cord injury.A reassessment of whether cortical motor neurons die following spinal cord injury.Epidermal neural crest stem cell (EPI-NCSC)--mediated recovery of sensory function in a mouse model of spinal cord injury.Peripheral nerve grafts after cervical spinal cord injury in adult cats.Diffuse traumatic axonal injury in the mouse induces atrophy, c-Jun activation, and axonal outgrowth in the axotomized neuronal populationA systematic review of directly applied biologic therapies for acute spinal cord injury.A surviving intact branch stabilizes remaining axon architecture after injury as revealed by in vivo imaging in the mouse spinal cord.Evaluation of Injured Axons Using Two-Photon Excited Fluorescence Microscopy after Spinal Cord Contusion Injury in YFP-H Line Mice.Cortical reorganization after spinal cord injury: always for good?Axonal regeneration induced by blockade of glial inhibitors coupled with activation of intrinsic neuronal growth pathwaysComprehensive Corticospinal Labeling with mu-crystallin Transgene Reveals Axon Regeneration after Spinal Cord Trauma in ngr1-/- Mice.Chondroitinase ABC promotes compensatory sprouting of the intact corticospinal tract and recovery of forelimb function following unilateral pyramidotomy in adult mice.Mild traumatic brain injury in the mouse induces axotomy primarily within the axon initial segmentChondroitinase enhances cortical map plasticity and increases functionally active sprouting axons after brain injuryEvidence for an Age-Dependent Decline in Axon Regeneration in the Adult Mammalian Central Nervous System.Combination of engineered Schwann cell grafts to secrete neurotrophin and chondroitinase promotes axonal regeneration and locomotion after spinal cord injury.Large-scale chondroitin sulfate proteoglycan digestion with chondroitinase gene therapy leads to reduced pathology and modulates macrophage phenotype following spinal cord contusion injury.Guidance molecules in axon regenerationFunctions of chondroitin sulfate and heparan sulfate in the developing brain.The glial scar in spinal cord injury and repair.Manipulating the extracellular matrix and its role in brain and spinal cord plasticity and repair.Delayed treatment with chondroitinase ABC promotes sensorimotor recovery and plasticity after stroke in aged rats.Efficient secretion of biologically active Chondroitinase ABC from mammalian cells in the absence of an N-terminal signal peptide.Modification of N-glycosylation sites allows secretion of bacterial chondroitinase ABC from mammalian cellsExpression of the regeneration-associated protein SPRR1A in primary sensory neurons and spinal cord of the adult mouse following peripheral and central injury.Trafficking and processing of bacterial proteins by mammalian cells: Insights from chondroitinase ABC.iPSC-derived neural precursor cells: potential for cell transplantation therapy in spinal cord injury.Heterogeneous spine loss in layer 5 cortical neurons after spinal cord injury.Cholinergic neuron-like cells derived from bone marrow stromal cells induced by tricyclodecane-9-yl-xanthogenate promote functional recovery and neural protection after spinal cord injury.The Biology of Regeneration Failure and Success After Spinal Cord Injury.
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P2860
The yellow fluorescent protein (YFP-H) mouse reveals neuroprotection as a novel mechanism underlying chondroitinase ABC-mediated repair after spinal cord injury.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
The yellow fluorescent protein ...... pair after spinal cord injury.
@ast
The yellow fluorescent protein ...... pair after spinal cord injury.
@en
type
label
The yellow fluorescent protein ...... pair after spinal cord injury.
@ast
The yellow fluorescent protein ...... pair after spinal cord injury.
@en
prefLabel
The yellow fluorescent protein ...... pair after spinal cord injury.
@ast
The yellow fluorescent protein ...... pair after spinal cord injury.
@en
P2093
P2860
P1476
The yellow fluorescent protein ...... pair after spinal cord injury.
@en
P2093
Lucy M Carter
Meirion Davies
Michelle L Starkey
Sonia F Akrimi
P2860
P304
14107-14120
P356
10.1523/JNEUROSCI.2217-08.2008
P407
P577
2008-12-01T00:00:00Z