BIM deficiency differentially impacts the function of kidney endothelial and epithelial cells through modulation of their local microenvironment
about
Cyp1b1 mediates periostin regulation of trabecular meshwork development by suppression of oxidative stressPro-apoptotic BIM is an essential initiator of physiological endothelial cell death independent of regulation by FOXO3.Aberrant production of extracellular matrix proteins and dysfunction in kidney endothelial cells with a short duration of diabetesOver-production of nitric oxide by oxidative stress-induced activation of the TGF-β1/PI3K/Akt pathway in mesangial cells cultured in high glucoseThe contribution of the programmed cell death machinery in innate immune cells to lupus nephritis.
P2860
BIM deficiency differentially impacts the function of kidney endothelial and epithelial cells through modulation of their local microenvironment
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
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2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
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2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
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name
BIM deficiency differentially ...... f their local microenvironment
@ast
BIM deficiency differentially ...... f their local microenvironment
@en
type
label
BIM deficiency differentially ...... f their local microenvironment
@ast
BIM deficiency differentially ...... f their local microenvironment
@en
prefLabel
BIM deficiency differentially ...... f their local microenvironment
@ast
BIM deficiency differentially ...... f their local microenvironment
@en
P2093
P2860
P1476
BIM deficiency differentially ...... f their local microenvironment
@en
P2093
Christine M Sorenson
Margaret E Morrison
Nader Sheibani
SunYoung Park
Zafer Gurel
P2860
P304
P356
10.1152/AJPRENAL.00498.2011
P577
2011-12-14T00:00:00Z