L-arginine availability regulates inducible nitric oxide synthase-dependent host defense against Helicobacter pylori.
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Arginine Metabolism in Bacterial Pathogenesis and Cancer TherapyCrystal Structure of Arginase from Plasmodium falciparum and Implications for l -Arginine Depletion in Malarial Infection,Difluoromethylornithine is a novel inhibitor of Helicobacter pylori growth, CagA translocation, and interleukin-8 inductionA novel virulence strategy for Pseudomonas aeruginosa mediated by an autotransporter with arginine-specific aminopeptidase activityActivation of the epidermal growth factor receptor in macrophages regulates cytokine production and experimental colitis.Modulation of the arginase pathway in the context of microbial pathogenesis: a metabolic enzyme moonlighting as an immune modulatorArginase II restricts host defense to Helicobacter pylori by attenuating inducible nitric oxide synthase translation in macrophagesHelicobacter pylori induces ERK-dependent formation of a phospho-c-Fos c-Jun activator protein-1 complex that causes apoptosis in macrophagesIncreased expression and cellular localization of spermine oxidase in ulcerative colitis and relationship to disease activityComparison of oxygen-induced radical intermediates in iNOS oxygenase domain with those from nNOS and eNOS.Cationic amino acid transporter 2 enhances innate immunity during Helicobacter pylori infection.Helicobacter pylori and gastric cancer: factors that modulate disease risk.L-arginine supplementation improves responses to injury and inflammation in dextran sulfate sodium colitisHeme oxygenase-1 dysregulates macrophage polarization and the immune response to Helicobacter pylori.Polyamines Impair Immunity to Helicobacter pylori by Inhibiting L-Arginine Uptake Required for Nitric Oxide ProductionHelicobacter pylori in the pathogenesis of gastric cancer and gastric lymphomaRole of innate immunity in Helicobacter pylori-induced gastric malignancy.The apolipoprotein E-mimetic peptide COG112 inhibits NF-kappaB signaling, proinflammatory cytokine expression, and disease activity in murine models of colitis.Immune evasion by Helicobacter pylori is mediated by induction of macrophage arginase IIHigh-throughput multi-analyte Luminex profiling implicates eotaxin-1 in ulcerative colitis.Liposome-encapsulated ISMN: a novel nitric oxide-based therapeutic agent against Staphylococcus aureus biofilms.Arginine and polyamines in Helicobacter pylori-induced immune dysregulation and gastric carcinogenesis.Berberine promotes recovery of colitis and inhibits inflammatory responses in colonic macrophages and epithelial cells in DSS-treated mice.Systems-wide analyses of mucosal immune responses to Helicobacter pylori at the interface between pathogenicity and symbiosisL-arginine uptake by cationic amino acid transporter 2 is essential for colonic epithelial cell restitution.TRPM2 ion channels regulate macrophage polarization and gastric inflammation during Helicobacter pylori infection.The L-Arginine Transporter Solute Carrier Family 7 Member 2 Mediates the Immunopathogenesis of Attaching and Effacing Bacteria.Expression of arginase I and inducible nitric oxide synthase in the peripheral blood and lymph nodes of HIV‑positive patients.Functional modularity of the arginine catabolic mobile element contributes to the success of USA300 methicillin-resistant Staphylococcus aureus.TCA cycle inactivation in Staphylococcus aureus alters nitric oxide production in RAW 264.7 cells.The apolipoprotein E-mimetic peptide COG112 inhibits the inflammatory response to Citrobacter rodentium in colonic epithelial cells by preventing NF-kappaB activation.Detrimental effects of Bartonella henselae are counteracted by L-arginine and nitric oxide in human endothelial progenitor cells.The Immune Battle against Helicobacter pylori Infection: NO Offense.Outer and inner membrane proteins compose an arginine-agmatine exchange system in Chlamydophila pneumoniaeDeletion of cationic amino acid transporter 2 exacerbates dextran sulfate sodium colitis and leads to an IL-17-predominant T cell responseDifferential impact of L-arginine deprivation on the activation and effector functions of T cells and macrophages.Pneumocystis mediates overexpression of antizyme inhibitor resulting in increased polyamine levels and apoptosis in alveolar macrophages.EGFR regulates macrophage activation and function in bacterial infection.Spermine oxidase is a regulator of macrophage host response to Helicobacter pylori: enhancement of antimicrobial nitric oxide generation by depletion of spermineParasitic antigens alter macrophage polarization during Schistosoma japonicum infection in mice.
P2860
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P2860
L-arginine availability regulates inducible nitric oxide synthase-dependent host defense against Helicobacter pylori.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
L-arginine availability regula ...... e against Helicobacter pylori.
@ast
L-arginine availability regula ...... e against Helicobacter pylori.
@en
type
label
L-arginine availability regula ...... e against Helicobacter pylori.
@ast
L-arginine availability regula ...... e against Helicobacter pylori.
@en
prefLabel
L-arginine availability regula ...... e against Helicobacter pylori.
@ast
L-arginine availability regula ...... e against Helicobacter pylori.
@en
P2093
P2860
P50
P356
P1476
L-arginine availability regula ...... e against Helicobacter pylori.
@en
P2093
Mohammad Asim
Preston Y Kim
Rupesh Chaturvedi
P2860
P304
P356
10.1128/IAI.00578-07
P407
P577
2007-06-11T00:00:00Z