Mitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failure
about
Catestatin increases the expression of anti-apoptotic and pro-angiogenetic factors in the post-ischemic hypertrophied heart of SHR.Functional and pharmacological characteristics of permeability transition in isolated human heart mitochondria.Cardiomyopathy and Worsened Ischemic Heart Failure in SM22-α Cre-Mediated Neuropilin-1 Null Mice: Dysregulation of PGC1α and Mitochondrial Homeostasis.Enhancing Hematopoietic Stem Cell Transplantation Efficacy by Mitigating Oxygen Shock.Proteotoxic stress and circulating cell stress proteins in the cardiovascular diseases.Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Deletion of Cyclophilin D Impairs β-Oxidation and Promotes Glucose MetabolismNanotechnology inspired tools for mitochondrial dysfunction related diseases.Selective Inhibition of the Mitochondrial Permeability Transition Pore Protects against Neurodegeneration in Experimental Multiple SclerosisCyclosporine A-nanoparticles enhance the therapeutic benefit of adipose tissue-derived stem cell transplantation in a swine myocardial infarction model.Cyclosporin A in left ventricular remodeling after myocardial infarction.DJ-1 protects against cell death following acute cardiac ischemia-reperfusion injury.Targeting mitochondria for cardioprotection: examining the benefit for patients.Peptidyl-prolyl isomerases: a full cast of critical actors in cardiovascular diseases.Small Interfering RNA Targeting Mitochondrial Calcium Uniporter Improves Cardiomyocyte Cell Viability in Hypoxia/Reoxygenation Injury by Reducing Calcium Overload.Mitochondrial cyclophilin D ablation is associated with the activation of Akt/p70S6K pathway in the mouse kidney.Inhibition of NAADP signalling on reperfusion protects the heart by preventing lethal calcium oscillations via two-pore channel 1 and opening of the mitochondrial permeability transition pore.Co-administration of the flavanol (-)-epicatechin with doxycycline synergistically reduces infarct size in a model of ischemia reperfusion injury by inhibition of mitochondrial swelling.Heart failure-induced activation of phospholipase iPLA2γ generates hydroxyeicosatetraenoic acids opening the mitochondrial permeability transition pore.Ablation of Cyclophilin D Results in an Activation of FAK, Akt, and ERK Pathways in the Mouse Heart.Cyclophilin D promotes tubular cell damage and the development of interstitial fibrosis in the obstructed kidney.Mitochondrial DNA as an inflammatory mediator in cardiovascular diseases.Histamine H2 receptor activation exacerbates myocardial ischemia/reperfusion injury by disturbing mitochondrial and endothelial function.Ripk3 promotes ER stress-induced necroptosis in cardiac IR injury: A mechanism involving calcium overload/XO/ROS/mPTP pathway.
P2860
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P2860
Mitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failure
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
@zh-cn
name
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@ast
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@en
type
label
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@ast
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@en
prefLabel
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@ast
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@en
P2093
P2860
P50
P1476
Mitochondrial cyclophilin-D as ...... rdial infarction heart failure
@en
P2093
Derek M Yellon
Sapna Arjun
Shiang Y Lim
P2860
P304
P356
10.1111/J.1582-4934.2010.01235.X
P577
2011-11-01T00:00:00Z