Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart. - Wikidata - awgldk - TriplyDB

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.

http://www.wikidata.org/entity/Q33180709

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Using proteomics to uncover extracellular matrix interactions during cardiac remodeling.A1 adenosine receptor overexpression attenuates ischemia-reperfusion-induced apoptosis and caspase 3 activity Pyridostigmine restores cardiac autonomic balance after small myocardial infarction in mice Thymosin-β4 prevents cardiac rupture and improves cardiac function in mice with myocardial infarction.Targeted deletion of apoptosis signal-regulating kinase 1 attenuates left ventricular remodeling.Mitigation of the progression of heart failure with sildenafil involves inhibition of RhoA/Rho-kinase pathway Cardiac remodeling and physical training post myocardial infarction Right ventricular dilatation after left ventricular acute myocardial infarction is predictive of extremely high peri-infarctual apoptosis at postmortem examination in humans Mechanisms of induction of adenosine receptor genes and its functional significance.Mitochondrial cyclophilin-D as a potential therapeutic target for post-myocardial infarction heart failure Adiponectin protects against the development of systolic dysfunction following myocardial infarction.Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice.Liposomal tetrahydrobiopterin preserves eNOS coupling in the post-ischemic heart conferring in vivo cardioprotection.Impact of mitral regurgitation on exercise capacity and clinical outcomes in patients with ischemic left ventricular dysfunction.Morphological aspects of apoptosis in heart diseases Short term triiodo-L-thyronine treatment inhibits cardiac myocyte apoptosis in border area after myocardial infarction in rats.Antiapoptotic effect of implanted embryonic stem cell-derived early-differentiated cells in aging rats after myocardial infarction.Sex-related differences in intrinsic myocardial properties influence cardiac function in middle-aged rats during infarction-induced left ventricular remodeling Toll-like receptor signaling: a critical modulator of cell survival and ischemic injury in the heart Cerebrospinal fluid pharmacology: an improved pharmacology approach for chinese herbal medicine research.Effect of carvedilol on cardiomyocyte apoptosis in a rat model of myocardial infarction: a role for toll-like receptor 4.Role of mitogen-activated protein kinase in cardiac hypertrophy and heart failure Apoptosis predominates in nonmyocytes in heart failure.Mechanisms of cell death in heart disease.Beta1-adrenergic receptors stimulate cardiac contractility and CaMKII activation in vivo and enhance cardiac dysfunction following myocardial infarction.Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.Induction of myocardial infarcts of a predictable size and location by branch pattern probability-assisted coronary ligation in C57BL/6 mice.New insights into mechanisms of cardioprotection mediated by thyroid hormones.Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution.Hippo pathway deficiency reverses systolic heart failure after infarction.Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction Traditional Chinese medicine Qili qiangxin inhibits cardiomyocyte apoptosis in rats following myocardial infarction.Cardiac macrophages and apoptosis after myocardial infarction: effects of central MR blockade.Mouse cardiac surgery: comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies.Clinical implications of apoptosis in hypertensive heart disease Practical guidelines for rigor and reproducibility in preclinical and clinical studies on cardioprotection

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P2860

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.

http://www.wikidata.org/entity/Q33180709

description

2000 nî lūn-bûn

@nan

2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի հուլիսին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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2000年论文

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name

Progressive left ventricular r ...... ial infarction in mouse heart.

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Progressive left ventricular r ...... ial infarction in mouse heart.

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Progressive left ventricular r ...... ial infarction in mouse heart.

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Progressive left ventricular r ...... ial infarction in mouse heart.

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Progressive left ventricular r ...... ial infarction in mouse heart.

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AJPHEART.2000.279.1.H422

P1433

American Journal of Physiology - Heart and Circulatory Physiology

P1476

Progressive left ventricular r ...... ial infarction in mouse heart.

@en

P2093

Amin J

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Apstein CS

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Chang DL

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Colucci WS

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Eberli FR

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Jain M

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P2860

The lethal effects of cytokine-induced nitric oxide on cardiac myocytes are blocked by nitric oxide synthase antagonism or transforming growth factor beta

Stretch-induced programmed myocyte cell death

Apopain/CPP32 cleaves proteins that are essential for cellular repair: a fundamental principle of apoptotic death.

Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes. Involvement of the sphingolipid signaling cascade in cardiac cell death.

Overexpression of insulin-like growth factor-1 in mice protects from myocyte death after infarction, attenuating ventricular dilation, wall stress, and cardiac hypertrophy.

Stretch-mediated release of angiotensin II induces myocyte apoptosis by activating p53 that enhances the local renin-angiotensin system and decreases the Bcl-2-to-Bax protein ratio in the cell.

Myocyte apoptosis during acute myocardial infarction in the mouse localizes to hypoxic regions but occurs independently of p53

Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats.

Ischaemic myocardial injury and ventricular remodelling.

Myocardial infarct size and ventricular function in rats.

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279

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2000-07-01T00:00:00Z

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10899082

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