Endogenous modifiers of cigarette smoke exposure within the lung.

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Chronic obstructive pulmonary disease Is periodontitis a comorbidity of COPD or can associations be explained by shared risk factors/behaviors?The activation of HMGB1 as a progression factor on inflammation response in normal human bronchial epithelial cells through RAGE/JNK/NF-κB pathway.

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P2860

Endogenous modifiers of cigarette smoke exposure within the lung.

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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Endogenous modifiers of cigarette smoke exposure within the lung.

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Endogenous modifiers of cigarette smoke exposure within the lung.

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Endogenous modifiers of cigarette smoke exposure within the lung.

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Endogenous modifiers of cigarette smoke exposure within the lung.

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A McGarry Houghton

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An immune basis for lung parenchymal destruction in chronic obstructive pulmonary disease and emphysema

Neutrophil elastase-mediated degradation of IRS-1 accelerates lung tumor growth

Api6/AIM/Spα/CD5L overexpression in alveolar type II epithelial cells induces spontaneous lung adenocarcinoma

Elastin fragments drive disease progression in a murine model of emphysema

Infiltration of neutrophils is required for acquisition of metastatic phenotype of benign murine fibrosarcoma cells: implication of inflammation-associated carcinogenesis and tumor progression.

Association of radiographic emphysema and airflow obstruction with lung cancer.

Alpha1-antitrypsin deficiency carriers, tobacco smoke, chronic obstructive pulmonary disease, and lung cancer risk

K-ras activation generates an inflammatory response in lung tumors.

Natural history and life expectancy in severe alpha1-antitrypsin deficiency, Pi Z.

Airways obstruction and the risk for lung cancer.

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66-68

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scholarly article

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10.1513/PATS.201108-046MS

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2012-05-01T00:00:00Z

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22550246

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3359106

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Endogenous modifiers of cigarette smoke exposure within the lung.

about

P2860

P2860

Endogenous modifiers of cigarette smoke exposure within the lung.

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