K-ras activation generates an inflammatory response in lung tumors. - Wikidata - awgldk - TriplyDB

K-ras activation generates an inflammatory response in lung tumors.

K-ras activation generates an inflammatory response in lung tumors.

http://www.wikidata.org/entity/Q40351368

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Contextual extracellular cues promote tumor cell EMT and metastasis by regulating miR-200 family expression The IL-8/IL-8R Axis: A Double Agent in Tumor Immune Resistance Neutrophils in Cancer: Two Sides of the Same Coin The CXCL8-CXCR1/2 pathways in cancer RAS oncogenes: weaving a tumorigenic web Neutrophil elastase-mediated degradation of IRS-1 accelerates lung tumor growth Peripheral blood immune cell methylation profiles are associated with nonhematopoietic cancers Crystalline silica-induced leukotriene B4-dependent inflammation promotes lung tumour growth Knockout of the tumor suppressor gene Gprc5a in mice leads to NF-kappaB activation in airway epithelium and promotes lung inflammation and tumorigenesis Phosphatidylinositol 3-kinase mediates bronchioalveolar stem cell expansion in mouse models of oncogenic K-ras-induced lung cancer Tobacco smoke promotes lung tumorigenesis by triggering IKKbeta- and JNK1-dependent inflammation.Expression signatures of metastatic capacity in a genetic mouse model of lung adenocarcinoma Sox2 is required for maintenance and differentiation of bronchiolar Clara, ciliated, and goblet cells.Keratin promoter based gene manipulation in the murine conducting airway Significance of baseline and change in neutrophil-to-lymphocyte ratio in predicting prognosis: a retrospective analysis in advanced pancreatic ductal adenocarcinoma.Mutant KRAS promotes malignant pleural effusion formation.Vanadium pentoxide induces pulmonary inflammation and tumor promotion in a strain-dependent manner.K-ras mutation targeted to gastric tissue progenitor cells results in chronic inflammation, an altered microenvironment, and progression to intraepithelial neoplasia.IDO is a nodal pathogenic driver of lung cancer and metastasis development.Innate inflammation induced by the 8-oxoguanine DNA glycosylase-1-KRAS-NF-κB pathway T helper 17 cells play a critical pathogenic role in lung cancer.Immune response in ovarian cancer: how is the immune system involved in prognosis and therapy: potential for treatment utilization.Lung adenocarcinomas induced in mice by mutant EGF receptors found in human lung cancers respond to a tyrosine kinase inhibitor or to down-regulation of the receptors.Interleukin 6, but not T helper 2 cytokines, promotes lung carcinogenesis.Prognostic significance of C-reactive protein polymorphism and KRAS/BRAF in synchronous liver metastasis from colorectal cancer.c-Myc regulates proliferation and Fgf10 expression in airway smooth muscle after airway epithelial injury in mouse.Design of hybrid multimodal poly(lactic-co-glycolic acid) polymer nanoparticles for neutrophil labeling, imaging and tracking.The association between expressions of Ras and CD68 in the angiogenesis of breast cancers.Whole transcriptome analysis reveals an 8-oxoguanine DNA glycosylase-1-driven DNA repair-dependent gene expression linked to essential biological processes The putative tumor suppressor gene EphA3 fails to demonstrate a crucial role in murine lung tumorigenesis or morphogenesis.Characterization of the cell of origin for small cell lung cancer Loss of RhoA Exacerbates, Rather Than Dampens, Oncogenic K-Ras Induced Lung Adenoma Formation in Mice.Respiratory Homeostasis and Exploitation of the Immune System for Lung Cancer Vaccines Identification of urine protein biomarkers with the potential for early detection of lung cancer.Direct and indirect roles for β-catenin in facultative basal progenitor cell differentiation Discovery of an oncogenic activity in p27Kip1 that causes stem cell expansion and a multiple tumor phenotype Expression of KRASG12V in Zebrafish Gills Induces Hyperplasia and CXCL8-Associated Inflammation.Cre-mediated recombination in mouse Clara cells.Deep Sequence Analysis of Non-Small Cell Lung Cancer: Integrated Analysis of Gene Expression, Alternative Splicing, and Single Nucleotide Variations in Lung Adenocarcinomas with and without Oncogenic KRAS Mutations Endogenous modifiers of cigarette smoke exposure within the lung.

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P2860

K-ras activation generates an inflammatory response in lung tumors.

http://www.wikidata.org/entity/Q40351368

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K-ras activation generates an inflammatory response in lung tumors.

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K-ras activation generates an inflammatory response in lung tumors.

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K-ras activation generates an inflammatory response in lung tumors.

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Bronson RT

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El-Bardeesy N

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Hezel A

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Houghton AM

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Ji H

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Marconcini LA

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P2860

Inflammation and cancer

Structure and neutrophil-activating properties of a novel inflammatory peptide (ENA-78) with homology to interleukin 8

Induction and apoptotic regression of lung adenocarcinomas by regulation of a K-Ras transgene in the presence and absence of tumor suppressor genes

Classification of human lung carcinomas by mRNA expression profiling reveals distinct adenocarcinoma subclasses

Identification of bronchioalveolar stem cells in normal lung and lung cancer

Thymocyte emigration is mediated by active movement away from stroma-derived factors

Biological implications of macrophage infiltration in human tumor angiogenesis.

MMP-9 supplied by bone marrow-derived cells contributes to skin carcinogenesis.

Early restriction of peripheral and proximal cell lineages during formation of the lung.

Interleukin-8 and the chemokine family.

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2105-2112

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10.1038/SJ.ONC.1209237

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14

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