TREM-1--expressing intestinal macrophages crucially amplify chronic inflammation in experimental colitis and inflammatory bowel diseases.
about
Inflammatory bowel disease related innate immunity and adaptive immunityDiversity of Intestinal Macrophages in Inflammatory Bowel DiseasesThe role of macrophages and dendritic cells in the initiation of inflammation in IBDThe triggering receptor expressed on myeloid cells (TREM) in inflammatory bowel disease pathogenesisAssociation among genetic predisposition, gut microbiota, and host immune response in the etiopathogenesis of inflammatory bowel diseaseDiagnostic value of soluble triggering receptor expressed on myeloid cells in paediatric sepsis: a systematic reviewHVEM signalling promotes colitisCombinations of host biomarkers predict mortality among Ugandan children with severe malaria: a retrospective case-control studyA distinct peripheral blood monocyte phenotype is associated with parasite inhibitory activity in acute uncomplicated Plasmodium falciparum malaria.Activation of the epidermal growth factor receptor in macrophages regulates cytokine production and experimental colitis.Activation of TGF-β activated kinase 1 promotes colon mucosal pathogenesis in inflammatory bowel disease.LPS independent activation of the pro-inflammatory receptor Trem1 by C/EBPε in granulocytesLong-term follow-up with Granulocyte and Monocyte Apheresis re-treatment in patients with chronically active inflammatory bowel disease.The linker for activation of B cells (LAB)/non-T cell activation linker (NTAL) regulates triggering receptor expressed on myeloid cells (TREM)-2 signaling and macrophage inflammatory responses independently of the linker for activation of T cells.The macrophage in HIV-1 infection: from activation to deactivation?Correlation between soluble triggering receptor expressed on myeloid cells-1 and endoscopic activity in intestinal Behçet's disease.A novel ligand-independent peptide inhibitor of TREM-1 suppresses tumor growth in human lung cancer xenografts and prolongs survival of mice with lipopolysaccharide-induced septic shock.Inflammation anergy in human intestinal macrophages is due to Smad-induced IkappaBalpha expression and NF-kappaB inactivation.Stromal down-regulation of macrophage CD4/CCR5 expression and NF-κB activation mediates HIV-1 non-permissiveness in intestinal macrophagesRelationship between expression of triggering receptor-1 on myeloid cells in intestinal tissue and intestinal barrier dysfunction in severe acute pancreatitis.TREM-1 as a potential therapeutic target in neonatal sepsis.Tacrolimus (FK506) suppresses TREM-1 expression at an early but not at a late stage in a murine model of fungal keratitisPerturbations of mucosal homeostasis through interactions of intestinal microbes with myeloid cells.Transcriptional analysis reveals gender-specific changes in the aging of the human immune system.Proteins and an inflammatory network expressed in colon tumors.Inactivation of DAP12 in PMN inhibits TREM1-mediated activation in rheumatoid arthritisFunctional specializations of intestinal dendritic cell and macrophage subsets that control Th17 and regulatory T cell responses are dependent on the T cell/APC ratio, source of mouse strain, and regional localization.TREM-1 amplifies corneal inflammation after Pseudomonas aeruginosa infection by modulating Toll-like receptor signaling and Th1/Th2-type immune responses.Correlation between triggering receptor expressed on myeloid cells-1 and clinical disease activity in Chinese patients with ulcerative colitis.The role of the macrophage in sentinel responses in intestinal immunity.TREM-2 promotes macrophage survival and lung disease after respiratory viral infectionCD14(+) macrophages that accumulate in the colon of African AIDS patients express pro-inflammatory cytokines and are responsive to lipopolysaccharide.TREM-1 Promotes Pancreatitis-Associated Intestinal Barrier Dysfunction.Targeting TREM-1 Signaling in the Presence of Antibiotics is Effective Against Streptococcal Toxic-Shock-Like Syndrome (STSLS) Caused by Streptococcus suisMicroglial malfunction: the third rail in the development of Alzheimer's disease.Profiles of microRNA networks in intestinal epithelial cells in a mouse model of colitis.Sensor molecules in intestinal innate immunity against bacterial infections.Unique CD14 intestinal macrophages contribute to the pathogenesis of Crohn disease via IL-23/IFN-gamma axis.TGR5 signalling inhibits the production of pro-inflammatory cytokines by in vitro differentiated inflammatory and intestinal macrophages in Crohn's diseaseChronic inflammation and cancer: emerging roles of triggering receptors expressed on myeloid cells.
P2860
Q26741407-BC891447-8C16-442C-9B09-CBD286F8BAA2Q26772342-65E87364-001E-4DE1-9798-425237C32326Q26827408-DC86D555-EE5C-46BE-A0DC-48A4DCE9E343Q26862713-3F49804C-DEF4-44EE-9B87-D278956839DFQ27006589-497A0D2E-02D5-43D6-8E5A-3FC8C9118164Q28076186-B3D79E44-2FA1-4557-A954-8E7919E77BF3Q28477792-E68E1172-1366-4132-ADEF-B70966C5BE9FQ28742367-83764A70-561F-4A96-8EAB-B025CFC4BFB0Q33511953-89450600-8147-432C-B86B-91C8581276E6Q33554130-D6AA745C-7AC2-4C57-81DA-26C215BA0241Q33567451-CDDBB1DA-4D8B-439B-83C5-2853D54E7923Q33601239-2E93104F-478E-4A02-99B1-57E5E9C392E9Q33626027-B2952BF1-FE2C-4D6B-843B-9A1FC453F246Q33661530-303F41F4-7540-4730-BFAF-906DF991B5A1Q33810779-D2992EB7-0222-4A63-B61E-288374BEFDA5Q33822597-B5196DAD-B93B-4F0D-8941-7D58DDD79FA3Q33869944-5134A8C0-D510-4F0C-A010-6B989EF5C167Q33911451-638ED262-4BD2-411E-A8BB-EA1932068509Q33921277-C6BA79FF-5258-4A04-A0C3-A30E2096287EQ34031855-64EFDF0E-CD87-477E-B044-458A9BAC5125Q34042162-D27B4CA9-C512-498A-948F-64DDC77632E3Q34612464-1E764BE3-E236-4BFE-9CFF-3FE92057E5D3Q34756285-F20E8A94-CE32-4F20-BA4A-8E238541585DQ34776449-91091A2D-80C1-4E99-BB68-E2408ABFF99DQ34958339-803E4A22-AF75-444D-BCC7-4D47F62485B1Q35038322-E4619571-A16A-4506-A6CE-9F79DFFC619DQ35091226-10AF85A5-28E4-4F98-82E7-6B9A8E9FC981Q35328809-8395734E-802C-4B11-A450-F53EC1828E3CQ35480033-7DD7EC7E-4808-4E12-8D87-D33143A6E7E7Q35545583-25A57540-7FDE-4CE5-94B0-EA581F0A4214Q35568471-3871B459-412F-4F91-BEBB-3DC7A841693FQ35810983-8C0E8895-8F61-4054-9E10-8DA43DF448BEQ35961344-E8168347-7807-45E5-9E7A-C917A4923C1FQ36271812-C2E2D785-B406-47DD-A4FC-A75370BA8EACQ36348184-BA1B243C-F407-4043-BA25-549876457244Q36357175-F7F38F89-1084-4377-B601-6896CA133BC3Q36388846-11399136-A3B5-402F-84C3-09BCC6DA7DBFQ36667276-10B73001-63BF-469E-89B7-9385A02C96BDQ36790557-EC5CE123-C05A-4223-9930-D5AF98B0474EQ36793296-1A459710-5F40-49BA-8C7C-F7DA1ACE1FF0
P2860
TREM-1--expressing intestinal macrophages crucially amplify chronic inflammation in experimental colitis and inflammatory bowel diseases.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@ast
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@en
type
label
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@ast
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@en
prefLabel
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@ast
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@en
P2860
P356
P1476
TREM-1--expressing intestinal ...... d inflammatory bowel diseases.
@en
P2093
Axel Bouchon
Frank Seibold
P2860
P304
P356
10.1172/JCI30602
P407
P577
2007-10-01T00:00:00Z