Dual Targeting of Akt and mTORC1 Impairs Repair of DNA Double-Strand Breaks and Increases Radiation Sensitivity of Human Tumor Cells.
about
Akt1 and Akt3 but not Akt2 through interaction with DNA-PKcs stimulate proliferation and post-irradiation cell survival of K-RAS-mutated cancer cells.Ataxia-Telangiectasia Mutated Modulation of Carbon Metabolism in Cancer.Combined inhibition of AKT and HSF1 suppresses breast cancer stem cells and tumor growth.Akt1 Stimulates Homologous Recombination Repair of DNA Double-Strand Breaks in a Rad51-Dependent Manner.Exploiting radiation-induced signaling to increase the susceptibility of resistant cancer cells to targeted drugs: AKT and mTOR inhibitors as an example.hTERT peptide fragment GV1001 demonstrates radioprotective and antifibrotic effects through suppression of TGF‑β signaling.Enhancing radiosensitivity of melanoma cells through very high dose rate pulses released by a plasma focus device.
P2860
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P2860
Dual Targeting of Akt and mTORC1 Impairs Repair of DNA Double-Strand Breaks and Increases Radiation Sensitivity of Human Tumor Cells.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@ast
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@en
type
label
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@ast
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@en
prefLabel
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@ast
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@en
P2093
P2860
P1433
P1476
Dual Targeting of Akt and mTOR ...... sitivity of Human Tumor Cells.
@en
P2093
Astrid Grottke
H Peter Rodemann
Julia Manes
Katharina Mueck
Mahmoud Toulany
Manfred Jücker
Marina Holler
P2860
P304
P356
10.1371/JOURNAL.PONE.0154745
P407
P577
2016-05-03T00:00:00Z