Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis - Wikidata - awgldk - TriplyDB

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

http://www.wikidata.org/entity/Q36022483

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EGFR inhibition attenuates diabetic nephropathy through decreasing ROS and endoplasmic reticulum stress.Signalling pathways involved in hypoxia-induced renal fibrosis.microRNA-153 Targets mTORC2 Component Rictor to Inhibit Glioma Cells Quercetin Inhibits Fibroblast Activation and Kidney Fibrosis Involving the Suppression of Mammalian Target of Rapamycin and β-catenin Signaling.Rapamycin Attenuates Cardiac Fibrosis in Experimental Uremic Cardiomyopathy by Reducing Marinobufagenin Levels and Inhibiting Downstream Pro-Fibrotic Signaling.Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and Fibrosis.PHF14: an innate inhibitor against the progression of renal fibrosis following folic acid-induced kidney injury.Activation of mTOR (mechanistic target of rapamycin) in rheumatic diseases.Omega-3 Polyunsaturated Fatty Acids Attenuate Fibroblast Activation and Kidney Fibrosis Involving MTORC2 Signaling Suppression.Roles of mTOR complexes in the kidney: implications for renal disease and transplantation.The SOD Mimic, MnTE-2-PyP, Protects from Chronic Fibrosis and Inflammation in Irradiated Normal Pelvic Tissues.Rictor/mammalian target of rapamycin complex 2 promotes macrophage activation and kidney fibrosis.Inhibiting aerobic glycolysis suppresses renal interstitial fibroblast activation and renal fibrosis.Development and Characterization of an In Vitro Model for Radiation-Induced Fibrosis.Wnt/β-Catenin-Promoted Macrophage Alternative Activation Contributes to Kidney Fibrosis.TGF-β and the Tissue Microenvironment: Relevance in Fibrosis and Cancer.Protein kinase C alpha drives fibroblast activation and kidney fibrosis by stimulating autophagic flux.

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Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

http://www.wikidata.org/entity/Q36022483

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

@zh-cn

name

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@ast

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@en

type

label

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@ast

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@en

prefLabel

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@ast

Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

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Kidney International

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Rictor/mTORC2 signaling mediates TGFβ1-induced fibroblast activation and kidney fibrosis

@en

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Chunsun Dai

http://www.w3.org/2001/XMLSchema#string

Jiafa Ren

http://www.w3.org/2001/XMLSchema#string

Jianzhong Li

http://www.w3.org/2001/XMLSchema#string

Junwei Yang

http://www.w3.org/2001/XMLSchema#string

Lei Jiang

http://www.w3.org/2001/XMLSchema#string

Weichun He

http://www.w3.org/2001/XMLSchema#string

Weiping Yuan

http://www.w3.org/2001/XMLSchema#string

Xin Liu

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P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Selective activation of Akt1 by mammalian target of rapamycin complex 2 regulates cancer cell migration, invasion, and metastasis

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

mTOR is a key modulator of ageing and age-related disease

Cellular and molecular mechanisms of fibrosis

Rictor/mTORC2 is essential for maintaining a balance between beta-cell proliferation and cell size

The mammalian target of rapamycin complex 2 controls folding and stability of Akt and protein kinase C

mTOR signaling at a glance

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis

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scholarly article

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10.1038/KI.2015.119

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3

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2015-05-13T00:00:00Z

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276361613

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4558569

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