Animal Models Are Valid to Uncover Disease Mechanisms.

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Animal Models Are Valid to Uncover Disease Mechanisms.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

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2016年论文

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2016年论文

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name

Animal Models Are Valid to Uncover Disease Mechanisms.

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Animal Models Are Valid to Uncover Disease Mechanisms.

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Item

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Animal Models Are Valid to Uncover Disease Mechanisms.

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Animal Models Are Valid to Uncover Disease Mechanisms.

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Animal Models Are Valid to Uncover Disease Mechanisms.

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Animal Models Are Valid to Uncover Disease Mechanisms.

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P1476

Animal Models Are Valid to Uncover Disease Mechanisms.

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P2093

Wolfgang Wurst

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P2860

Reciprocal Effects on Neurocognitive and Metabolic Phenotypes in Mouse Models of 16p11.2 Deletion and Duplication Syndromes

Mouse genomic variation and its effect on phenotypes and gene regulation

Behavioral abnormalities and circuit defects in the basal ganglia of a mouse model of 16p11.2 deletion syndrome.

Dosage-dependent phenotypes in models of 16p11.2 lesions found in autism

Highly efficient Cas9-mediated transcriptional programming

Intact-Brain Analyses Reveal Distinct Information Carried by SNc Dopamine Subcircuits

One-step generation of mice carrying reporter and conditional alleles by CRISPR/Cas-mediated genome engineering.

Copy number variation in the dosage-sensitive 16p11.2 interval accounts for only a small proportion of autism incidence: a systematic review and meta-analysis.

Spatiotemporal 16p11.2 protein network implicates cortical late mid-fetal brain development and KCTD13-Cul3-RhoA pathway in psychiatric diseases.

P304

e1006013

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scholarly article

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10.1371/JOURNAL.PGEN.1006013

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P433

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P478

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2016-05-26T00:00:00Z

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27227339

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4881889

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