Spatiotemporal 16p11.2 protein network implicates cortical late mid-fetal brain development and KCTD13-Cul3-RhoA pathway in psychiatric diseases.
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Prenatal Neurogenesis in Autism Spectrum DisordersMechanisms for spatiotemporal regulation of Rho-GTPase signaling at synapsesIdentification of the BRD1 interaction network and its impact on mental disorder riskGenome-wide prediction and functional characterization of the genetic basis of autism spectrum disorderA highly penetrant form of childhood apraxia of speech due to deletion of 16p11.2.Integrative analysis of genetic data sets reveals a shared innate immune component in autism spectrum disorder and its co-morbiditiesDelineating the Common Biological Pathways Perturbed by ASD's Genetic Etiology: Lessons from Network-Based StudiesAnimal Models Are Valid to Uncover Disease Mechanisms.Post-transcriptional regulation of SHANK3 expression by microRNAs related to multiple neuropsychiatric disordersDiffusion Tensor Imaging Provides Evidence of Possible Axonal Overconnectivity in Frontal Lobes in Autism Spectrum Disorder Toddlers.Dysregulation of miRNA-9 in a Subset of Schizophrenia Patient-Derived Neural Progenitor CellsThe Cellular and Molecular Landscapes of the Developing Human Central Nervous System.Reversal of dendritic phenotypes in 16p11.2 microduplication mouse model neurons by pharmacological targeting of a network hub.Synaptic Actin Dysregulation, a Convergent Mechanism of Mental Disorders?Comprehensive Analyses of Tissue-Specific Networks with Implications to Psychiatric Diseases.Bacurd1/Kctd13 and Bacurd2/Tnfaip1 are interacting partners to Rnd proteins which influence the long-term positioning and dendritic maturation of cerebral cortical neurons.Application of induced pluripotent stem cells to understand neurobiological basis of bipolar disorder and schizophrenia.A cullin 4B-RING E3 ligase complex fine-tunes pancreatic δ cell paracrine interactions.Ubiquitylation at the crossroads of development and disease.Kctd13 deletion reduces synaptic transmission via increased RhoA.Integrated Bayesian analysis of rare exonic variants to identify risk genes for schizophrenia and neurodevelopmental disorders.The 16p11.2 homologs fam57ba and doc2a generate certain brain and body phenotypes.Vitamin D-related genes are subjected to significant de novo mutation burdens in autism spectrum disorder.Circuitry-Based Human Neuroanatomy for the Next Generation in Psychiatry and Neuroscience.Landscape of Conditional eQTL in Dorsolateral Prefrontal Cortex and Co-localization with Schizophrenia GWAS.Loss of the Chr16p11.2 ASD candidate gene leads to aberrant neuronal differentiation in the SH-SY5Y neuronal cell model
P2860
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P2860
Spatiotemporal 16p11.2 protein network implicates cortical late mid-fetal brain development and KCTD13-Cul3-RhoA pathway in psychiatric diseases.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@en
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@nl
type
label
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@en
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@nl
prefLabel
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@en
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@nl
P2093
P2860
P50
P1433
P1476
Spatiotemporal 16p11.2 protein ...... thway in psychiatric diseases.
@en
P2093
David E Hill
Guan Ning Lin
Irma Lemmens
Jan Tavernier
Marc Vidal
Roser Corominas
P2860
P304
P356
10.1016/J.NEURON.2015.01.010
P407
P577
2015-02-01T00:00:00Z